文献检索，用中文搜 PubMed

Pilocarpine and other cholinomimetic drugs convert isoprenaline to a vasoconstrictor and pressor agent.2. This effect of pilocarpine was abolished by atropine; it is thus an acetylcholine-like response. It was not dependent on the integrity of the central nervous system or the adrenal glands and was not abolished by ganglionic blockade.3. The constrictor action of isoprenaline after pilocarpine was abolished by propranolol; this action of isoprenaline is thus on the beta-adrenoceptor. Another beta-adrenoceptor stimulating agent, salbutamol, resembled isoprenaline in this situation, though papaverine and acetylcholine did not.4. The constrictor action of isoprenaline after pilocarpine was abolished by phenoxybenzamine, guanethidine and cocaine; the effect did not appear after reserpine pretreatment.5. These results suggest an action of cholinomimetic drugs at adrenergic nerve endings which permits the uptake of beta-adrenoceptor stimulating agents resulting in the release of neuronal transmitter.

毛果芸香碱及其他拟胆碱药可使异丙肾上腺素转变为血管收缩剂和升压剂。
毛果芸香碱的这一作用可被阿托品消除；因此这是一种类似乙酰胆碱的反应。它不依赖于中枢神经系统或肾上腺的完整性，且不被神经节阻断所消除。
毛果芸香碱作用后异丙肾上腺素的收缩作用可被普萘洛尔消除；因此异丙肾上腺素的这一作用是通过β-肾上腺素能受体。在这种情况下，另一种β-肾上腺素能受体激动剂沙丁胺醇与异丙肾上腺素相似，而罂粟碱和乙酰胆碱则不然。
毛果芸香碱作用后异丙肾上腺素的收缩作用可被酚苄明、胍乙啶和可卡因消除；利血平预处理后该效应未出现。
这些结果提示拟胆碱药作用于肾上腺素能神经末梢，使β-肾上腺素能受体激动剂得以摄取，从而导致神经递质释放。

Pilocarpine and other cholinomimetic drugs convert isoprenaline to a vasoconstrictor and pressor agent.2. This effect of pilocarpine was abolished by atropine; it is thus an acetylcholine-like response. It was not dependent on the integrity of the central nervous system or the adrenal glands and was not abolished by ganglionic blockade.3. The constrictor action of isoprenaline after pilocarpine was abolished by propranolol; this action of isoprenaline is thus on the beta-adrenoceptor. Another beta-adrenoceptor stimulating agent, salbutamol, resembled isoprenaline in this situation, though papaverine and acetylcholine did not.4. The constrictor action of isoprenaline after pilocarpine was abolished by phenoxybenzamine, guanethidine and cocaine; the effect did not appear after reserpine pretreatment.5. These results suggest an action of cholinomimetic drugs at adrenergic nerve endings which permits the uptake of beta-adrenoceptor stimulating agents resulting in the release of neuronal transmitter.

毛果芸香碱及其他拟胆碱药可使异丙肾上腺素转变为血管收缩剂和升压剂。
毛果芸香碱的这一作用可被阿托品消除；因此这是一种类似乙酰胆碱的反应。它不依赖于中枢神经系统或肾上腺的完整性，且不被神经节阻断所消除。
毛果芸香碱作用后异丙肾上腺素的收缩作用可被普萘洛尔消除；因此异丙肾上腺素的这一作用是通过β-肾上腺素能受体。在这种情况下，另一种β-肾上腺素能受体激动剂沙丁胺醇与异丙肾上腺素相似，而罂粟碱和乙酰胆碱则不然。
毛果芸香碱作用后异丙肾上腺素的收缩作用可被酚苄明、胍乙啶和可卡因消除；利血平预处理后该效应未出现。
这些结果提示拟胆碱药作用于肾上腺素能神经末梢，使β-肾上腺素能受体激动剂得以摄取，从而导致神经递质释放。

Suppr 超能文献