β-丙内酯对仙台病毒的作用。
Effect of -propiolactone on Sendai virus.
作者信息
Wainberg M A, Hjorth R N, Howe C
出版信息
Appl Microbiol. 1971 Oct;22(4):618-21. doi: 10.1128/am.22.4.618-621.1971.
Abstract
The biological properties (infectivity, hemagglutination, hemolysis, cell fusion, neuraminidase) of Sendai virus were dissociated on the basis of sensitivity to beta-propiolactone, by freeze-thawing, by heating at different temperatures, and by adsorption-elution with formalinized chicken erythrocytes. Possible mechanisms whereby beta-propiolactone selectively destroys viral infectivity are discussed.
摘要
仙台病毒的生物学特性（感染性、血凝、溶血、细胞融合、神经氨酸酶）基于对β-丙内酯的敏感性、通过冻融、在不同温度下加热以及用甲醛化鸡红细胞进行吸附洗脱而被分离。文中讨论了β-丙内酯选择性破坏病毒感染性的可能机制。
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