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Hepatic necrosis caused by furosemide.

作者信息

Mitchell J R, Potter W Z, Hinson J A, Jollow D J

出版信息

Nature. 1974 Oct 11;251(5475):508-11. doi: 10.1038/251508a0.

DOI:10.1038/251508a0
PMID:4424638
Abstract
摘要

相似文献

1
Hepatic necrosis caused by furosemide.速尿引起的肝坏死。
Nature. 1974 Oct 11;251(5475):508-11. doi: 10.1038/251508a0.
2
Humanized thymidine kinase-NOG mice can be used to identify drugs that cause animal-specific hepatotoxicity: a case study with furosemide.人源化胸苷激酶-NOG小鼠可用于鉴定引起动物特异性肝毒性的药物:以呋塞米为例的案例研究。
J Pharmacol Exp Ther. 2015 Jul;354(1):73-8. doi: 10.1124/jpet.115.224493. Epub 2015 May 11.
3
Metabolic activation of furosemide to a chemically reactive, hepatotoxic metabolite.
J Pharmacol Exp Ther. 1976 Oct;199(1):41-52.
4
Furosemide induced hepatotoxicity.呋塞米诱发的肝毒性。
J Pathol. 1981 Dec;135(4):301-14. doi: 10.1002/path.1711350407.
5
[Acute lesions of the proximal tubular epithelium of the rat kidney following a single high dosage of furosemide].[单次高剂量速尿后大鼠肾脏近端肾小管上皮的急性病变]
Klin Wochenschr. 1971 Dec 1;49(23):1298-300. doi: 10.1007/BF01733087.
6
[Experimental liver damage by furosemide for studying drugs with hepato-protective activity].[通过速尿诱导实验性肝损伤以研究具有肝保护活性的药物]
Eksp Med Morfol. 1989;28(3):47-50.
7
Nrf2 protects against furosemide-induced hepatotoxicity.Nrf2 可预防速尿引起的肝毒性。
Toxicology. 2014 Oct 3;324:35-42. doi: 10.1016/j.tox.2014.02.008. Epub 2014 May 6.
8
[Reaction of the hepatic tissue of various laboratory animals to single administration of thioacetamide].[不同实验动物肝脏组织对单次给予硫代乙酰胺的反应]
Beitr Pathol Anat. 1969;139(4):421-38.
9
Massive liver cell necrosis induced in the pig with carbon tetrachloride.用四氯化碳在猪身上诱导出大量肝细胞坏死。
S Afr Med J. 1974 Jun 12;48(28):1201-4.
10
Intraperitoneal administration of high doses of polyethylene glycol (PEG) causes hepatic subcapsular necrosis and low-grade peritonitis with a rise in hepatic biomarkers.腹腔内给予大剂量聚乙二醇(PEG)可导致肝包膜下坏死和低度腹膜炎,并使肝生物标志物升高。
Toxicology. 2013 Dec 15;314(2-3):262-6. doi: 10.1016/j.tox.2013.06.003. Epub 2013 Jul 3.

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IL-6 mediates the hepatic acute phase response after prerenal azotemia in a clinically defined murine model.白细胞介素 6 在临床定义的鼠模型肾前氮血症后介导肝脏急性期反应。
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Computational Approach to Structural Alerts: Furans, Phenols, Nitroaromatics, and Thiophenes.结构警示的计算方法:呋喃、酚类、硝基芳烃和噻吩
Chem Res Toxicol. 2017 Apr 17;30(4):1046-1059. doi: 10.1021/acs.chemrestox.6b00336. Epub 2017 Mar 14.
6
Humanized thymidine kinase-NOG mice can be used to identify drugs that cause animal-specific hepatotoxicity: a case study with furosemide.人源化胸苷激酶-NOG小鼠可用于鉴定引起动物特异性肝毒性的药物:以呋塞米为例的案例研究。
J Pharmacol Exp Ther. 2015 Jul;354(1):73-8. doi: 10.1124/jpet.115.224493. Epub 2015 May 11.
7
The role of the c-Jun N-terminal kinases 1/2 and receptor-interacting protein kinase 3 in furosemide-induced liver injury.c-Jun氨基末端激酶1/2及受体相互作用蛋白激酶3在速尿诱导的肝损伤中的作用
Xenobiotica. 2015 May;45(5):442-9. doi: 10.3109/00498254.2014.986250. Epub 2014 Nov 25.
8
Reactive metabolites in the biotransformation of molecules containing a furan ring.含呋喃环分子的生物转化中的反应性代谢物。
Chem Res Toxicol. 2013 Jan 18;26(1):6-25. doi: 10.1021/tx3003824. Epub 2012 Oct 24.
9
Biliary excretion of glutathione and glutathione disulfide in the rat. Regulation and response to oxidative stress.大鼠体内谷胱甘肽和谷胱甘肽二硫化物的胆汁排泄。氧化应激的调节与反应。
J Clin Invest. 1984 Jan;73(1):124-33. doi: 10.1172/JCI111182.
10
The role of biotransformation in chemical-induced liver injury.生物转化在化学性肝损伤中的作用。
Environ Health Perspect. 1976 Jun;15:27-38. doi: 10.1289/ehp.761527.