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Impaired drug metabolism in rats associated with acute inflammation: a possible assay for anti-injury agents.

作者信息

Beck F J, Whitehouse M W

出版信息

Proc Soc Exp Biol Med. 1974 Jan;145(1):135-40. doi: 10.3181/00379727-145-37763.

DOI:10.3181/00379727-145-37763
PMID:4544268
Abstract
摘要

相似文献

1
Impaired drug metabolism in rats associated with acute inflammation: a possible assay for anti-injury agents.
Proc Soc Exp Biol Med. 1974 Jan;145(1):135-40. doi: 10.3181/00379727-145-37763.
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Impaired drug metabolism in rats with adjuvant-induced arthritis: a brief review.
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The diphasic action of halothane on the oxidative metabolism of drugs by the liver. An in-vitro study in the rat.
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Effect of hypoxia alone or combined with inflammation and 3-methylcholanthrene on hepatic cytochrome P450 in conscious rabbits.单独缺氧或缺氧联合炎症及3-甲基胆蒽对清醒家兔肝细胞色素P450的影响。
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Selective effect of adjuvant arthritis on the disposition of propranolol enantiomers in rats detected using a stereospecific HPLC assay.使用立体特异性高效液相色谱法检测佐剂性关节炎对大鼠体内普萘洛尔对映体处置的选择性作用。
Pharm Res. 1993 Feb;10(2):294-9. doi: 10.1023/a:1018907431893.
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Effects of acute and chronic inflammation on the pharmacokinetics of prednisolone in rats.
急性和慢性炎症对大鼠体内泼尼松龙药代动力学的影响。
Pharm Res. 1994 Apr;11(4):541-4. doi: 10.1023/a:1018966516195.
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Additional inhibitors of rat serum acute phase alpha 2-macroglobulin levels. Effect of 6-mercaptopurine and some lipoxygenase and cyclo-oxygenase inhibitors.大鼠血清急性期α2-巨球蛋白水平的其他抑制剂。6-巯基嘌呤及一些脂氧合酶和环氧化酶抑制剂的作用。
Agents Actions. 1982 Jul;12(3):340-3. doi: 10.1007/BF01965400.
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Effects of acute febrile infectious diseases on the oral pharmacokinetics and effects of nitrendipine enantiomers and of bisoprolol.
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Alteration of Hepatic microsomal enzyme systems and the lethal action of non-steroidal anti-arthritic drugs in acute and chronic models of inflammation.肝微粒体酶系统的改变以及非甾体类抗炎药在急慢性炎症模型中的致死作用。
Agents Actions. 1977 Jul;7(2):289-97. doi: 10.1007/BF01969987.
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Depression of drug metabolism in the mouse by a combination of Mycobacterium butyricum and anaesthetics.丁酸分枝杆菌与麻醉剂联合作用对小鼠药物代谢的抑制作用。
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