在有空气存在的情况下，黑光（325至400纳米）对流感嗜血杆菌的致死和诱变作用。
Lethal and mutagenic action of black light (325 to 400 nm) on Haemophilus influenzae in the presence of air.
作者信息
Cabrera-Juarez E, Espinosa-Lara M
出版信息
J Bacteriol. 1974 Mar;117(3):960-4. doi: 10.1128/jb.117.3.960-964.1974.
Abstract
Near-ultraviolet (UV) light (325 to 400 nm), in the presence of air and the absence of exogenous photosensitizing compounds, is lethal and mutagenic for Haemophilus influenzae. The lethal effect is the same for both wild type and streptomycin-resistant mutants, indicating that the mutants are not selected by the irradiation. The inactivation and mutagenicity show a large shoulder, suggesting the existence of repair systems. Filters were used to eliminate the possibility of short-UV irradiation. The effective radiation is between 325 to 400 nm. The lethal and mutagenic effects are higher during mid and late log phase than during early log or stationary phase.
摘要
近紫外光（UV，325至400纳米），在有空气且不存在外源性光敏化合物的情况下，对流感嗜血杆菌具有致死性和致突变性。野生型和链霉素抗性突变体的致死效应相同，这表明突变体不是由辐射选择产生的。失活和致突变性呈现出一个大的“肩区”，表明存在修复系统。使用滤光片以排除短波长紫外线照射的可能性。有效辐射范围在325至400纳米之间。对数中期和后期的致死和致突变效应高于对数早期或稳定期。
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