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The relationship of intracellular pathways of iron metabolism to cellular iron overload and the iron storage diseases. Cell sap and cytocavitary network pathways in relation to lysosomal storage and turnover of iron macromolecules.

作者信息

Trump B F, Valigorsky J M, Arstila A U, Mergner W J, Kinney T D

出版信息

Am J Pathol. 1973 Aug;72(2):295-336.

PMID:4579280
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1903989/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/c9c1af653bcb/amjpathol00252-0206-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/3146281aced7/amjpathol00252-0199-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/353a57e9a790/amjpathol00252-0199-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/ec49568fb249/amjpathol00252-0199-c.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/b038d0469e6a/amjpathol00252-0199-d.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/8ecab0e700d9/amjpathol00252-0200-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/3cbbfe1f0abc/amjpathol00252-0201-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/187d2b342b15/amjpathol00252-0201-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/b6ca0a2c3731/amjpathol00252-0202-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/6ce95ab1198f/amjpathol00252-0203-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/e6b3f7e315b8/amjpathol00252-0195-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/f6917c9622e6/amjpathol00252-0204-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/71b9be988224/amjpathol00252-0171-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/c8fbede6099f/amjpathol00252-0196-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/f78ea3f438e5/amjpathol00252-0205-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/d276ad805b3a/amjpathol00252-0197-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/94760c2a6681/amjpathol00252-0197-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/c9c1af653bcb/amjpathol00252-0206-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/3146281aced7/amjpathol00252-0199-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/353a57e9a790/amjpathol00252-0199-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/ec49568fb249/amjpathol00252-0199-c.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/b038d0469e6a/amjpathol00252-0199-d.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/8ecab0e700d9/amjpathol00252-0200-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/3cbbfe1f0abc/amjpathol00252-0201-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/187d2b342b15/amjpathol00252-0201-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/b6ca0a2c3731/amjpathol00252-0202-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/6ce95ab1198f/amjpathol00252-0203-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/e6b3f7e315b8/amjpathol00252-0195-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/f6917c9622e6/amjpathol00252-0204-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/71b9be988224/amjpathol00252-0171-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/c8fbede6099f/amjpathol00252-0196-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/f78ea3f438e5/amjpathol00252-0205-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/d276ad805b3a/amjpathol00252-0197-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/94760c2a6681/amjpathol00252-0197-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ddb/1903989/c9c1af653bcb/amjpathol00252-0206-a.jpg

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FAILURE OF ACTINOMYCIN D TO PREVENT INDUCTION OF LIVER APOFERRITIN AFTER IRON ADMINISTRATION.放线菌素D未能阻止铁给药后肝脏脱铁铁蛋白的诱导。
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GOLGI APPARATUS AND LYSOSOMES.高尔基体和溶酶体。
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THE ROLE OF THE INTESTINE IN IRON KINETICS.肠道在铁动力学中的作用。
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Liver iron transport.肝脏铁转运
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Hemosiderin granules: cytotoxic response to arsenic exposure in channel catfish.
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Cytochemical studies on the ferritin-containing vesicles of the rat incisor ameloblasts with special reference to the acid phosphatase activity.
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Phagocytotic and iron-storing capacities of stromal cells in the rat endometrium. A histochemical and ultrastructural study.大鼠子宫内膜基质细胞的吞噬和铁储存能力。一项组织化学和超微结构研究。
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Biliary excretion of iron from hepatocyte lysosomes in the rat. A major excretory pathway in experimental iron overload.大鼠肝细胞溶酶体中铁的胆汁排泄。实验性铁过载中的一条主要排泄途径。
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PROTEIN SYNTHESIS, STORAGE, AND DISCHARGE IN THE PANCREATIC EXOCRINE CELL. AN AUTORADIOGRAPHIC STUDY.胰腺外分泌细胞中的蛋白质合成、储存与释放。一项放射自显影研究。
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ON FERRITIN AND ITS PRODUCTION BY CELLS GROWING.关于铁蛋白及其由正在生长的细胞产生的情况。
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IRON STORAGE IN N-2-FLUORENYLACETAMIDE-INDUCED HEPATIC INJURY. ELECTRON MICROSCOPIC OBSERVATIONS FOLLOWING THE INJECTION OF IRON-DEXTRAN.N-2-芴基乙酰胺诱导的肝损伤中的铁储存。注射右旋糖酐铁后的电子显微镜观察
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EFFECT OF HEPATIC INJURIES UPON IRON STORAGE IN THE LIVER.肝脏损伤对肝脏铁储存的影响。
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INTESTINAL MUCOSAL MECHANISMS CONTROLLING IRON ABSORPTION.控制铁吸收的肠道黏膜机制
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Swelling and lysis of rat liver mitochondria induced by ferrous ions.亚铁离子诱导大鼠肝线粒体肿胀和裂解。
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