大肠杆菌K-12 tolE突变体的遗传学与生理学特性以及半乳糖对其表型的抑制作用
Genetics and physiology of a tolE mutant of Escherichia coli K-12 and phenotypic suppression of its phenotype by galactose.
作者信息
Eriksson-Grennberg K G, Nordström K
出版信息
J Bacteriol. 1973 Sep;115(3):1219-22. doi: 10.1128/jb.115.3.1219-1222.1973.
Abstract
The tolE mutation causes tolerance to colicins E2 and E3 as well as other effects on the phenotype of Escherichia coli K-12. The lipopolysaccharide of the mutant shows a reduction in the content of galactose, glucose, and rhamnose. The phenotype of the mutant, including the composition of the lipopolysaccharide, is suppressed by galactose. The map position is shown by the gene order trp-purB-tolE-tolD-galKETO.
摘要
tolE突变导致对大肠杆菌素E2和E3产生耐受性，以及对大肠杆菌K-12的表型产生其他影响。突变体的脂多糖中半乳糖、葡萄糖和鼠李糖的含量有所减少。突变体的表型，包括脂多糖的组成，会被半乳糖抑制。其图谱位置由基因顺序trp-purB-tolE-tolD-galKETO表示。
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