T2 噬菌体空壳感染大肠杆菌后 30S 核糖体亚基出现缺陷。
Defective 30S ribosomal subunits after infection of Escherichia coli by T2 ghosts.
作者信息
Simon M, Kennell D
出版信息
J Virol. 1974 Nov;14(5):1310-3. doi: 10.1128/JVI.14.5.1310-1313.1974.
Abstract
Ribosomes from ghost-treated Escherichia coli can form in vitro initiation complexes but cannot translate added template RNA because of a defect in the 30S subunit; 16S rRNA is degraded.
摘要
经空壳处理的大肠杆菌核糖体能够在体外形成起始复合物，但由于30S亚基存在缺陷，无法翻译添加的模板RNA；16S rRNA已降解。
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本文引用的文献
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Inhibition of host protein synthesis during infection of Escherichi coli by bacteriophage T4. 3. Inhibition by ghosts.噬菌体T4感染大肠杆菌期间宿主蛋白质合成的抑制作用。3. 空衣壳的抑制作用。
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