[第二次四氯化碳中毒对四氯化碳损伤小鼠肝脏的影响] - Suppr

Suppr

超能文献

[Effect of a second CCl 4 -intoxication on CCl 4 -damaged mouse liver].

作者信息

Gerhard H, Schultze B, Maurer W

出版信息

Virchows Arch B Cell Pathol. 1972;10(3):184-99.

PMID:4624425

Abstract

摘要

相似文献

[Effect of a second CCl 4 -intoxication on CCl 4 -damaged mouse liver].[第二次四氯化碳中毒对四氯化碳损伤小鼠肝脏的影响]

Virchows Arch B Cell Pathol. 1972;10(3):184-99.

[Autoradiographic investigation of proliferation and polyploidization during CCl4-induced liver regeneration in mice (author's transl)].四氯化碳诱导小鼠肝再生过程中增殖与多倍体化的放射自显影研究（作者译）

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[Cytological and morphometric investigation of mouse liver regeneration after CCl4 intoxication (author's transl)].四氯化碳中毒后小鼠肝脏再生的细胞学与形态测量学研究（作者译）

Virchows Arch B Cell Pathol. 1973 Dec 17;14(4):345-59.

[Histoautoradiographic studies on liver regeneration after carbon tetrachloride poisoning].[四氯化碳中毒后肝脏再生的组织放射自显影研究]

Med Welt. 1972 Sep 23;23(39):1348-51.

[Liver regeneration in mice following prolonged administration of CCl-4].[四氯化碳长期给药后小鼠的肝脏再生]

Biull Eksp Biol Med. 1973 Aug;76(8):112-4.

[Proliferation kinetics of bile duct epithelia in the regenerating mouse liver after CCl4-poisoning].[四氯化碳中毒后再生小鼠肝脏中胆管上皮细胞的增殖动力学]

Virchows Arch B Cell Pathol. 1975;17(3):213-27.

[Radioautographic study of the relation of the rhythm of reparative liver regeneration to the rhythm of the pathogenic factor].[放射性自显影研究修复性肝再生节律与致病因素节律的关系]

Biull Eksp Biol Med. 1972 May;73(5):113-5.

An autoradiographic study of protein synthesis in mouse liver parenchymal cells during CC14 intoxication.四氯化碳中毒时小鼠肝脏实质细胞蛋白质合成的放射自显影研究

Am J Pathol. 1969 Jan;54(1):73-82.

[Quantitative model of mouse liver regeneration after CCl4 poisoning (author's transl)].四氯化碳中毒后小鼠肝脏再生的定量模型（作者译）

Virchows Arch B Cell Pathol. 1973 Dec 17;14(4):361-71.

Acute liver toxicity by carbon tetrachloride in HSP70 knock out mice.热休克蛋白70基因敲除小鼠中四氯化碳引起的急性肝毒性。

Exp Toxicol Pathol. 2007 Sep;59(1):29-34. doi: 10.1016/j.etp.2007.02.005. Epub 2007 Jun 20.

引用本文的文献

Amplified interactive toxicity of chemicals at nontoxic levels: mechanistic considerations and implications to public health.化学物质在无毒水平下的增强交互毒性：机制考量及其对公众健康的影响

Environ Health Perspect. 1994 Nov;102 Suppl 9(Suppl 9):139-49. doi: 10.1289/ehp.94102s9139.

Increase in liver cell nuclear size after chronic cadmium treatment.慢性镉处理后肝细胞细胞核大小增加。

Arch Toxicol. 1981 Sep;48(2-3):209-11. doi: 10.1007/BF00310490.

Reduction of carbon tetrachloride toxicity by prior administration of a single small dose in mice and rats.在小鼠和大鼠中预先给予单次小剂量可降低四氯化碳的毒性。

Br J Exp Pathol. 1975 Apr;56(2):172-9.

Lanthanon-induced hepatotoxicity and its prevention by pretreatment with the same lanthanon.稀土元素诱导的肝毒性及其通过相同稀土元素预处理的预防作用。

Arch Toxicol. 1979 Dec;43(2):105-14. doi: 10.1007/BF00333617.

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