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An autoradiographic study of protein synthesis in mouse liver parenchymal cells during CC14 intoxication.

作者信息

Monlux G, Smuckler E A

出版信息

Am J Pathol. 1969 Jan;54(1):73-82.

PMID:5762433
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC2013364/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a4f5/2013364/ba8d56b9fd06/amjpathol00428-0096-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a4f5/2013364/b4f9fe94ef47/amjpathol00428-0093-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a4f5/2013364/ed1eb15c5985/amjpathol00428-0093-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a4f5/2013364/c962555d0157/amjpathol00428-0094-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a4f5/2013364/e7ac958913ea/amjpathol00428-0094-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a4f5/2013364/4f75d2440947/amjpathol00428-0095-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a4f5/2013364/540dd06b6e45/amjpathol00428-0095-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a4f5/2013364/f37032a02087/amjpathol00428-0096-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a4f5/2013364/ba8d56b9fd06/amjpathol00428-0096-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a4f5/2013364/b4f9fe94ef47/amjpathol00428-0093-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a4f5/2013364/ed1eb15c5985/amjpathol00428-0093-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a4f5/2013364/c962555d0157/amjpathol00428-0094-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a4f5/2013364/e7ac958913ea/amjpathol00428-0094-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a4f5/2013364/4f75d2440947/amjpathol00428-0095-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a4f5/2013364/540dd06b6e45/amjpathol00428-0095-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a4f5/2013364/f37032a02087/amjpathol00428-0096-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a4f5/2013364/ba8d56b9fd06/amjpathol00428-0096-b.jpg

相似文献

1
An autoradiographic study of protein synthesis in mouse liver parenchymal cells during CC14 intoxication.四氯化碳中毒时小鼠肝脏实质细胞蛋白质合成的放射自显影研究
Am J Pathol. 1969 Jan;54(1):73-82.
2
[An autoradiographic study of DNA and RNA synthesis in the liver cells of mice with chronic carbon tetrachloride poisoning].
Biull Eksp Biol Med. 1973 Apr;75(4):34-6.
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[On the mechanism of inhibition of protein synthesis in rat liver 30 minutes after CCl-4 damage].[关于四氯化碳损伤后30分钟大鼠肝脏蛋白质合成抑制机制的研究]
Acta Biol Med Ger. 1965;15(6):667-79.
4
[Autoradiographic studies of protein and nucleic acid metabolism of the liver after CC14 poisoning].
Acta Histochem. 1968:Suppl 8:315+.
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[Autoradiographic study of the disorder of protein synthesis in the mouse liver following CCL4 intoxication. Dose-effect graphs for reversible and irreversible damages].[四氯化碳中毒后小鼠肝脏蛋白质合成紊乱的放射自显影研究。可逆性和不可逆性损伤的剂量效应图]
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[Autoradiographic study of RNA synthesis of the liver during CCl4 intoxication].
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[Changes in amide nitrogen and urea nitrogen in the normal and CCl 4-intoxicated, isolated and perfused liver].[正常及四氯化碳中毒的离体灌注肝脏中酰胺氮和尿素氮的变化]
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[Electron autoradiographic studies of biosynthetic processes in the cell nucleus].[细胞核中生物合成过程的电子放射自显影研究]
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引用本文的文献

1
Early hypomethylation of 2'-O-ribose moieties in hepatocyte cytoplasmic ribosomal RNA underlies the protein synthetic defect produced by CCl4.四氯化碳导致的蛋白质合成缺陷的基础是肝细胞胞质核糖体核糖核酸中2'-O-核糖部分的早期低甲基化。
J Cell Biol. 1987 Aug;105(2):705-11. doi: 10.1083/jcb.105.2.705.

本文引用的文献

1
Cellular necrosis in the liver induced and modified by drugs.药物诱导和改变的肝脏细胞坏死。
Int Rev Exp Pathol. 1965;4:127-57.