抑制剂对正常人细胞和着色性干皮病细胞在X射线和紫外线照射后DNA修复的影响。 - Suppr

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超能文献

Effects of inhibitors on repair of DNA in normal human and xeroderma pigmentosum cells after exposure to x-rays and ultraviolet irradiation.

作者信息

Kleijer W J, Hoeksema J L, Sluyter M L, Bootsma D

出版信息

Mutat Res. 1973 Mar;17(3):385-94. doi: 10.1016/0027-5107(73)90010-9.

DOI:10.1016/0027-5107(73)90010-9

PMID:4631741

Abstract

摘要

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Effects of inhibitors on repair of DNA in normal human and xeroderma pigmentosum cells after exposure to x-rays and ultraviolet irradiation.抑制剂对正常人细胞和着色性干皮病细胞在X射线和紫外线照射后DNA修复的影响。

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Xeroderma pigmentosum variants have a slow recovery of DNA synthesis after irradiation with ultraviolet light.着色性干皮病变异型在紫外线照射后DNA合成恢复缓慢。

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DNA chain elongation and joining in normal human and xeroderma pigmentosum cells after ultraviolet irradiation.紫外线照射后正常人细胞和着色性干皮病细胞中的DNA链延伸与连接

Biophys J. 1972 Sep;12(9):1183-91. doi: 10.1016/S0006-3495(72)86154-X.

Xeroderma pigmentosum variant heterozygotes show reduced levels of recovery of replicative DNA synthesis in the presence of caffeine after ultraviolet irradiation.着色性干皮病变异型杂合子在紫外线照射后，于咖啡因存在的情况下，显示出复制性DNA合成的恢复水平降低。

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Nature. 1975 Dec 25;258(5537):760-3. doi: 10.1038/258760a0.

The influence of caffeine on cell survival in excision-proficient and excision-deficient xeroderma pigmentosum and normal human cell strains following ultraviolet-light irradiation.咖啡因对紫外线照射后切除修复功能正常、切除修复功能缺陷的着色性干皮病及正常人细胞株细胞存活的影响。

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Xeroderma pigmentosum cells with normal levels of excision repair have a defect in DNA synthesis after UV-irradiation.具有正常切除修复水平的着色性干皮病细胞在紫外线照射后DNA合成存在缺陷。

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DNA single-strand breaks during repair of UV damage in human fibroblasts and abnormalities of repair in xeroderma pigmentosum.人类成纤维细胞紫外线损伤修复过程中的DNA单链断裂及着色性干皮病的修复异常

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Different rate-limiting steps in excision repair of ultraviolet- and N-acetoxy-2-acetylaminofluorene-damaged DNA in normal human fibroblasts.

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