Kung C, Naito Y
Science. 1973 Jan 12;179(4069):195-6. doi: 10.1126/science.179.4069.195.
"Pawn," a genic mutant of Paramecium aurelia, cannot swim backward as the wild type can upon proper stimulation. In contrast, after membrane disruption by Triton X-100, the adenosine triphosphate-magnesium reactivated models of Pawns swim backward in the presence of calcium as wild-type models do. Thus, the mutant phenotype is due to an impairment in the membrane and not in the calcium-sensitive motile system.
“兵卒”是一种金色草履虫的基因变异体，在适当刺激下，它不能像野生型那样向后游动。相比之下，在用曲拉通X-100破坏细胞膜后，“兵卒”的三磷酸腺苷-镁再激活模型在有钙存在的情况下能像野生型模型一样向后游动。因此，突变表型是由于细胞膜受损，而非钙敏感运动系统受损。
