Giacchino J, Horwitz B A, Horowitz J M
J Appl Physiol Respir Environ Exerc Physiol. 1979 Jun;46(6):1049-53. doi: 10.1152/jappl.1979.46.6.1049.
Unrestrained rats were exposed to cold for 1 h during and immediately after exposure to hypergravic fields (1.5--4 G) to determine if they recover their ability to thermoregulate on reentry to 1-G conditions. In contrast to the decreased body temperatures observed when cold exposure occurred concurrently with acceleration, hypothalamic, carotid, and brown fat temperatures did not fall when rats were exposed to cold immediately after return to 1 G. These results support the hypothesis that the thermoregulatory alterations seen under hypergravic conditions are manifestations of an effect of ongoing exposure to hypergravity and can be reversed on termination of acceleration. The reversibility of the thermoregulatory impairment is apparently unaffected by the magnitude of the acceleration field over a range of 1.5--4 G.
将未加束缚的大鼠在暴露于超重重力场（1.5 - 4G）期间及暴露后立即置于寒冷环境中1小时，以确定它们在重新回到1G条件时是否恢复体温调节能力。与在加速过程中同时进行冷暴露时观察到的体温降低相反，当大鼠回到1G后立即暴露于寒冷环境时，下丘脑、颈动脉和棕色脂肪的温度并未下降。这些结果支持了以下假设：在超重重力条件下观察到的体温调节改变是持续暴露于超重重力的一种效应表现，并且在加速终止后可以逆转。在1.5 - 4G范围内，体温调节损伤的可逆性显然不受加速场强度的影响。
