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Loss of 7-methylguanine from rat liver DNA after methylation in vivo with methylmethanesulphonate or dimethylnitrosamine.

作者信息

Margison G P, Capps M J, O'Connor P J, Craig A W

出版信息

Chem Biol Interact. 1973 Feb;6(2):119-24. doi: 10.1016/0009-2797(73)90078-1.

DOI:10.1016/0009-2797(73)90078-1
PMID:4698159
Abstract
摘要

相似文献

1
Loss of 7-methylguanine from rat liver DNA after methylation in vivo with methylmethanesulphonate or dimethylnitrosamine.大鼠肝脏DNA在体内经甲磺酸甲酯或二甲基亚硝胺甲基化后7-甲基鸟嘌呤的丢失
Chem Biol Interact. 1973 Feb;6(2):119-24. doi: 10.1016/0009-2797(73)90078-1.
2
Differences in the patterns of methylation in rat liver ribosomal ribonucleic acid after reaction in vivo with methyl methanesulphonate and NN-dimethylnitrosamine.大鼠肝脏核糖体核糖核酸在体内与甲磺酸甲酯和N,N-二甲基亚硝胺反应后甲基化模式的差异。
Biochem J. 1972 Sep;129(3):519-28. doi: 10.1042/bj1290519.
3
The stability of rat liver ribonucleic acid in vivo after methylation with methyl methanesulphonate or dimethylnitrosamine.用甲磺酸甲酯或二甲基亚硝胺甲基化后大鼠肝脏核糖核酸在体内的稳定性
Biochem J. 1971 Dec;125(3):821-7. doi: 10.1042/bj1250821.
4
Methylation of nucleic acids and urinary excretion of 14 C-labeled 7-methylguanine by rats and man after administration of 4(5)-(3,3-dimethyl-1-triazeno)-imidazole5(4)-carboxomide.给予4(5)-(3,3-二甲基-1-三氮烯)-咪唑5(4)-甲酰胺后,大鼠和人体对核酸的甲基化作用及14C标记的7-甲基鸟嘌呤的尿排泄情况。
Toxicol Appl Pharmacol. 1971 Mar;18(3):707-19. doi: 10.1016/s0041-008x(71)80026-1.
5
Comparative studies of the hepatocarcinogen N,N-dimethylnitrosamine in vivo: reaction sites in rat liver DNA and the significance of their relative stabilities.体内肝癌致癌物N,N-二甲基亚硝胺的比较研究:大鼠肝脏DNA中的反应位点及其相对稳定性的意义。
Br J Cancer. 1973 Feb;27(2):153-66. doi: 10.1038/bjc.1973.19.
6
Probable methylation of nucleic acids of mouse colon by 1,2-dimethylhydrazine in vivo.1,2 - 二甲基肼在体内可能对小鼠结肠核酸进行甲基化作用。
Biochem Pharmacol. 1972 Feb 1;21(3):432-3. doi: 10.1016/0006-2952(72)90359-0.
7
Occurrence of 7-methylguanine in nucleic acids of rat liver.大鼠肝脏核酸中7-甲基鸟嘌呤的存在情况。
Biochem J. 1968 Mar;107(2):179-84. doi: 10.1042/bj1070179.
8
Rapid loss of 7-methylguanine from liver nucleic acids in mice during the initial stage of liver carcinogenesis induced by dimethylnitrosamine.在二甲基亚硝胺诱导的小鼠肝癌发生初始阶段,肝脏核酸中7-甲基鸟嘌呤迅速丢失。
Biochem Biophys Res Commun. 1974 May 7;58(1):242-9. doi: 10.1016/0006-291x(74)90918-8.
9
The pattern of methylated purines formed in DNA of intact and regenerating liver of rats treated with the carcinogen dimethylnitrosamine.用致癌物二甲基亚硝胺处理的大鼠完整肝脏和再生肝脏DNA中形成的甲基化嘌呤模式。
Biochim Biophys Acta. 1973 Jun 23;312(2):202-10. doi: 10.1016/0005-2787(73)90365-1.
10
[Reduced toxicity of dimethylnitrosamine in rats and mice after treatment with disulfiram].[用双硫仑治疗后大鼠和小鼠二甲基亚硝胺毒性降低]
Arzneimittelforschung. 1971 Oct;21(10):1560-2.

引用本文的文献

1
DNA-Protein Cross-Link Formation in Nucleosome Core Particles Treated with Methyl Methanesulfonate.用甲基甲磺酸处理核小体核心颗粒中的 DNA-蛋白质交联形成。
Chem Res Toxicol. 2019 Oct 21;32(10):2144-2151. doi: 10.1021/acs.chemrestox.9b00314. Epub 2019 Sep 18.
2
Histone tails decrease N7-methyl-2'-deoxyguanosine depurination and yield DNA-protein cross-links in nucleosome core particles and cells.组蛋白尾部可减少核小体核心颗粒和细胞中 N7-甲基-2'-脱氧鸟嘌呤的脱嘌呤作用,并产生 DNA-蛋白质交联。
Proc Natl Acad Sci U S A. 2018 Nov 27;115(48):E11212-E11220. doi: 10.1073/pnas.1813338115. Epub 2018 Nov 14.
3
Mutational specificity of depurination.
脱嘌呤的突变特异性
Proc Natl Acad Sci U S A. 1984 Mar;81(5):1494-8. doi: 10.1073/pnas.81.5.1494.
4
Urinary excretion of methylated purines following inhalation of dimethyl sulphate.吸入硫酸二甲酯后甲基化嘌呤的尿排泄情况。
Experientia. 1974 Jun 15;30(6):641-2. doi: 10.1007/BF01921519.
5
Persistence of O6-ethylguanine in rat-brain DNA: correlation with nervous system-specific carcinogenesis by ethylnitrosourea.大鼠脑DNA中O6-乙基鸟嘌呤的持久性:与乙基亚硝基脲诱导的神经系统特异性致癌作用的相关性。
Proc Natl Acad Sci U S A. 1974 Mar;71(3):639-43. doi: 10.1073/pnas.71.3.639.
6
Molecular and cellular mechanisms associated with pulse-carcinogenesis in the rat nerbous system by ethyinitrosourea: ethylation of nucleic acids and elimination rates of ethylated bases from the DNA of different tissues.乙基亚硝基脲诱导大鼠神经系统脉冲致癌作用的分子和细胞机制:核酸的乙基化及不同组织DNA中乙基化碱基的消除速率
Z Krebsforsch Klin Onkol Cancer Res Clin Oncol. 1974;82(1):37-64. doi: 10.1007/BF00304382.
7
Role of apurinic sites in the resistance of methylated oligodeoxyribonucleotides to degradation by spleen exonuclease.脱嘌呤位点在甲基化寡脱氧核糖核苷酸抵抗脾脏外切核酸酶降解中的作用。
Biochem J. 1975 Nov;151(2):249-56. doi: 10.1042/bj1510249.
8
Methylated purines in the deoxyribonucleic acid of various Syrian-golden-hamster tissues after administration of a hepatocarcinogenic dose of dimethylnitrosamine.给予致癌剂量的二甲基亚硝胺后,叙利亚金仓鼠各种组织的脱氧核糖核酸中的甲基化嘌呤。
Biochem J. 1976 Sep 1;157(3):627-34. doi: 10.1042/bj1570627.
9
Accumulation of O6-methylguanine in non-target-tissue deoxyribonucleic acid during chronic administration of dimethylnitrosamine.慢性给予二甲基亚硝胺期间非靶组织脱氧核糖核酸中O6-甲基鸟嘌呤的蓄积
Biochem J. 1977 Sep 1;165(3):463-8. doi: 10.1042/bj1650463.
10
Formation and subsequent removal of O6-methylguanine from deoxyribonucleic acid in rat liver and kidney after small doses of dimethylnitrosamine.小剂量二甲基亚硝胺作用后大鼠肝脏和肾脏中脱氧核糖核酸O6-甲基鸟嘌呤的形成及随后的去除
Biochem J. 1978 Sep 1;173(3):739-48. doi: 10.1042/bj1730739.