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Irreversible binding of 14C from 14CCl4 to liver microsomal lipids and proteins from rats pretreated with compounds altering microsomal mixed-function oxygenase activity.

作者信息

Díaz Gómez M I, Castro J A, de Ferreyra E C, D'Acosta N, de Castro C R

出版信息

Toxicol Appl Pharmacol. 1973 Aug;25(4):534-41. doi: 10.1016/0041-008x(73)90022-7.

DOI:10.1016/0041-008x(73)90022-7
PMID:4741774
Abstract
摘要

相似文献

1
Irreversible binding of 14C from 14CCl4 to liver microsomal lipids and proteins from rats pretreated with compounds altering microsomal mixed-function oxygenase activity.用改变微粒体混合功能氧化酶活性的化合物预处理大鼠后，四氯化碳中的14C与肝微粒体脂质和蛋白质的不可逆结合。
Toxicol Appl Pharmacol. 1973 Aug;25(4):534-41. doi: 10.1016/0041-008x(73)90022-7.
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Effect of drug pretreatment on carbon tetrachloride-induced lipid peroxidation in rat liver microsomal lipids.药物预处理对四氯化碳诱导的大鼠肝微粒体脂质过氧化的影响。
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4
Carbon tetrachloride target lipids in the rat liver microsomes. Effect of cystamine administration on their pattern of labeling by 14CCl4.四氯化碳对大鼠肝微粒体中的脂质产生作用。给予半胱胺对其被四氯化碳标记的模式的影响。
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Effects of ethynyl estradiol on liver microsomal mixed function oxygenase activity in male rats.乙炔雌二醇对雄性大鼠肝脏微粒体混合功能氧化酶活性的影响。
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Formation of irreversible protein-binding metabolites during microsomal metabolism of 14C-bromobenzene and 14C-bromophenol.
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Hydroperoxide catalyzed liver microsomal aromatic hydroxylation reactions involving cytochrome P-450.
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The effect of metyrapone on cellular respiration and microsomal drug oxidation.甲吡酮对细胞呼吸及微粒体药物氧化的作用。
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Late preventive effects of quinacrine on carbon tetrachloride induced liver necrosis.
Arch Toxicol. 1993;67(6):386-91. doi: 10.1007/BF01977399.
3
Decreased incorporation of 14C-leucine in different liver nuclear protein fractions at early stages of carbon tetrachloride poisoning in the rat.在大鼠四氯化碳中毒早期，不同肝细胞核蛋白组分中14C-亮氨酸的掺入减少。
Arch Toxicol. 1994;68(3):206-9. doi: 10.1007/s002040050056.
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Pyrazole prevention of CC14-induced ultrastructural changes in rat liver.吡唑对四氯化碳诱导的大鼠肝脏超微结构变化的预防作用。
Br J Exp Pathol. 1980 Oct;61(5):505-11.
5
Studies on the mechanism of enhancement of carbon tetrachloride hepatotoxicity by Triton WR 1339.吐温WR 1339增强四氯化碳肝毒性机制的研究。
Br J Exp Pathol. 1981 Oct;62(5):512-8.
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Studies on the mechanism of alloxan-diabetes potentiation of carbon tetrachloride-induced liver necrosis.四氧嘧啶糖尿病增强四氯化碳诱导的肝坏死机制的研究。
Br J Exp Pathol. 1982 Aug;63(4):388-93.
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Binding of trichloromethyl radicals to lipids of the hepatic endoplasmic reticulum during tetrachloromethane metabolism.四氯化碳代谢过程中三氯甲基自由基与肝脏内质网脂质的结合。
Biochem J. 1984 Nov 1;223(3):577-86. doi: 10.1042/bj2230577.
8
Carbon tetrachloride-induced early biochemical alterations but not necrosis in pigeon's liver.
Agents Actions. 1984 Oct;15(3-4):463-6. doi: 10.1007/BF01972389.
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Effect of inhibitors of the FAD-containing monooxygenase system from rat liver microsomes on monomethylhydrazine metabolism and activation to reactive metabolites.大鼠肝脏微粒体中含黄素单加氧酶系统抑制剂对单甲基肼代谢及活化为反应性代谢产物的影响。
Arch Toxicol. 1986 May;59(1):64-6. doi: 10.1007/BF00263961.
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Interaction of benznidazole reactive metabolites with nuclear and kinetoplastic DNA, proteins and lipids from Trypanosoma cruzi.苯硝唑反应性代谢产物与克氏锥虫的核及动基体DNA、蛋白质和脂质的相互作用。
Experientia. 1988 Oct 15;44(10):880-1. doi: 10.1007/BF01941187.