Dawkins R L, Zilko P J
Lancet. 1975 Jan 25;1(7900):200-2. doi: 10.1016/s0140-6736(75)91364-1.
Human polymyositis and aspects of myasthenia gravis may be consequences of subtle immunodeficiency states. Autoimmune processes leading to inflammatory muscle disease and the presence of associated tumours may reflect the partial loss of antibody-mediated homoeostatic mechanisms.
人类多发性肌炎和重症肌无力的某些方面可能是细微免疫缺陷状态的后果。导致炎性肌肉疾病的自身免疫过程以及相关肿瘤的存在可能反映了抗体介导的稳态机制的部分丧失。