红细胞δ-氨基乙酰丙酸脱水酶活性和血原卟啉浓度作为铅暴露和血红素生物合成改变的指标。 - Suppr

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超能文献

Erythrocyte delta-aminolaevulinic acid dehydratase activity and blood protoporphyrin concentrations as indices of lead exposure and altered haem biosynthesis.

作者信息

Meredith P A, Moore M R, Goldberg A

出版信息

Clin Sci (Lond). 1979 Jan;56(1):61-9. doi: 10.1042/cs0560061.

DOI:10.1042/cs0560061

PMID:477184

Abstract

摘要

相似文献

Erythrocyte delta-aminolaevulinic acid dehydratase activity and blood protoporphyrin concentrations as indices of lead exposure and altered haem biosynthesis.红细胞δ-氨基乙酰丙酸脱水酶活性和血原卟啉浓度作为铅暴露和血红素生物合成改变的指标。

Clin Sci (Lond). 1979 Jan;56(1):61-9. doi: 10.1042/cs0560061.

An evaluation of the use of haem-biosynthetic parameters in the detection of industrial and environmental lead exposure: erythrocyte delta-aminolaevulinate dehydratase and blood protoporphyrin concentrations. [proceedings].

Biochem Soc Trans. 1979 Feb;7(1):39-41. doi: 10.1042/bst0070039.

Erythrocyte delta-aminolaevulinic acid dehydratase activity and changes in delta-aminolaevulinic acid concentration in various forms of anaemia.各种贫血类型中红细胞δ-氨基-γ-酮戊酸脱水酶活性及δ-氨基-γ-酮戊酸浓度的变化

Br J Haematol. 1978 Nov;40(3):397-400. doi: 10.1111/j.1365-2141.1978.tb05811.x.

[Erythrocyte delta-aminolevulinic dehydratase and protoporphyrin in lead exposure].[铅暴露中的红细胞δ-氨基乙酰丙酸脱水酶和原卟啉]

Rev Med Chil. 1982 May;110(5):439-43.

Levels of delta-aminolevulinate dehydratase, uroporphyrinogen-I synthase, and protoporphyrin IX in erythrocytes from anemic mutant mice.贫血突变小鼠红细胞中δ-氨基乙酰丙酸脱水酶、尿卟啉原-I合酶和原卟啉IX的水平。

Proc Natl Acad Sci U S A. 1977 Mar;74(3):1181-4. doi: 10.1073/pnas.74.3.1181.

Increased erythrocyte uroporphyrinogen-l-synthetase, delta-aminolevulinic acid dehydratase and protoporphyrin in hemolytic anemias.溶血性贫血时红细胞尿卟啉原 -1- 合成酶、δ-氨基乙酰丙酸脱水酶及原卟啉增加。

Am J Med. 1977 Sep;63(3):359-64. doi: 10.1016/0002-9343(77)90273-x.

Zinc protoporphyrin in the erythrocytes of patients with lead intoxication and iron deficiency anemia.铅中毒和缺铁性贫血患者红细胞中的锌原卟啉。

Science. 1974 Dec 6;186(4167):936-8. doi: 10.1126/science.186.4167.936.

[Use of some indicators of "effect" in subjects exposed to lead].[铅暴露受试者中某些“效应”指标的应用]

Med Lav. 1978 Jan-Feb;69(1):96-7.

History and background of protoporphyrin testing.

Clin Chem. 1977 Feb;23(2 PT. 1):256-9.

Free erythrocyte protoporphyrin as an indicator of the biological effect of lead in adult males. II. Comparison between free erythrocyte protoporphyrin and other indicators of effect.游离红细胞原卟啉作为成年男性铅生物学效应的指标。II. 游离红细胞原卟啉与其他效应指标的比较。

Int Arch Occup Environ Health. 1976 Jun 3;37(2):89-105. doi: 10.1007/BF00378056.

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Animals (Basel). 2023 May 17;13(10):1670. doi: 10.3390/ani13101670.

Tissue distribution and urinary excretion of essential elements in rats orally exposed to aluminum chloride.口服氯化铝的大鼠体内必需元素的组织分布和尿排泄情况

Biol Trace Elem Res. 1991 Nov;31(2):131-8. doi: 10.1007/BF02990421.

Evaluation of lead exposure in workers at a lead-acid battery factory in Korea: with focus on activity of erythrocyte pyrimidine 5'-nucleotidase (P5N).韩国一家铅酸电池厂工人铅暴露的评估：重点关注红细胞嘧啶5'-核苷酸酶（P5N）的活性。

Occup Environ Med. 1995 Jul;52(7):484-8. doi: 10.1136/oem.52.7.484.

The in vivo effects of zinc on erythrocyte delta-aminolaevulinic acid dehydratase in man.

Int Arch Occup Environ Health. 1980 Feb;45(2):163-8. doi: 10.1007/BF01274135.

Persistent protoporphyrinemia in hereditary porphobilinogen synthase (delta-aminolevulinic acid dehydrase) deficiency under low lead exposure. A new molecular basis for the pathogenesis of lead intoxication.低铅暴露下遗传性胆色素原合酶（δ-氨基-γ-酮戊酸脱水酶）缺乏所致的持续性原卟啉血症。铅中毒发病机制的一种新分子基础。

Klin Wochenschr. 1982 Jun 15;60(12):599-606. doi: 10.1007/BF01711435.

Acute lead poisoning in inherited porphobilinogen synthase (delta-aminolevulinic acid dehydrase) deficiency.遗传性胆色素原合酶（δ-氨基乙酰丙酸脱水酶）缺乏症中的急性铅中毒

Blut. 1982 Aug;45(2):131-9. doi: 10.1007/BF00319941.

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