Vitamin A compounds and analogs as inhibitors of mixed-function oxidases that metabolize carcinogenic polycyclic hydrocarbons and other compounds.

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作者信息

Hill D L, Shih T W

出版信息

Cancer Res. 1974 Mar;34(3):564-70.

PMID:4812263

Abstract

摘要

相似文献

Vitamin A compounds and analogs as inhibitors of mixed-function oxidases that metabolize carcinogenic polycyclic hydrocarbons and other compounds.

Cancer Res. 1974 Mar;34(3):564-70.

Microsomal hydroxylases: induction and role in polycyclic hydrocarbon carcinogenesis and toxicity.微粒体羟化酶：在多环烃致癌作用和毒性中的诱导及作用

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Metabolism of polycyclic hydrocarbons by rat-lung preparations.大鼠肺组织制剂对多环烃的代谢

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Calculation of reactivity indices for benz(a)anthracene and benzo(a)pyrene and their approximative models of complex with enzyme epoxidase.苯并(a)蒽和苯并(a)芘及其与环氧化酶复合物的近似模型的反应活性指数计算。

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Metabolism of polycyclic aromatic hydrocarbons in cell cultuires.细胞培养中多环芳烃的代谢

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K-region epoxides of polycyclic hydrocarbons: formation and further metabolism by rat-lung preparations.

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Carcinogenic polynuclear hydrocarbons bind to macromolecules in cultured human bronchi.

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[Electrochemiluminescence of carcinogenic polycyclic hydrocarbons].[致癌多环芳烃的电化学发光]

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Binding of benzo(a)pyrene and dibenz(a,h)anthracene to the Ah receptor in mouse and rat hepatic cytosols.苯并(a)芘和二苯并(a,h)蒽与小鼠和大鼠肝细胞溶质中芳烃受体的结合。

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Anti-cancer action of retinoids.维甲酸的抗癌作用。

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Modulation of 3-methylcholanthrene toxicity in cultured neoplastic keratinocytes by glucocorticoids and retinoids is not accounted for by macromolecular adduct formation.糖皮质激素和类视黄醇对培养的肿瘤性角质形成细胞中3-甲基胆蒽毒性的调节作用并非由大分子加合物的形成所致。

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Induction of benzo[a]pyrene Mono-oxygenase in liver cell culture by the photochemical generation of active oxygen species. Evidence for the involvement of singlet oxygen and the formation of a stable inducing intermediate.通过光化学产生活性氧物种诱导肝细胞培养物中的苯并[a]芘单加氧酶。单线态氧参与及稳定诱导中间体形成的证据。

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Vitamin A (retinol) status in the rat after a single oral dose of 2,3,7,8-tetrachlorodibenzo-p-dioxin.单次口服2,3,7,8-四氯二苯并对二噁英后大鼠体内的维生素A（视黄醇）状态

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Vitamin A and the susceptibility of respiratory tract tissues to carcinogenic insult.维生素A与呼吸道组织对致癌损伤的易感性

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