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Proceedings: Potentiation by prostaglandins of the nociceptive activity of bradykinin in the dog knee joint.

作者信息

Ferreira S H, Moncada S, Vane J R

出版信息

Br J Pharmacol. 1974 Mar;50(3):461P.

PMID:4853840
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1776662/
Abstract
摘要

相似文献

1
Proceedings: Potentiation by prostaglandins of the nociceptive activity of bradykinin in the dog knee joint.论文:前列腺素对狗膝关节中缓激肽伤害性活动的增强作用
Br J Pharmacol. 1974 Mar;50(3):461P.
2
Proceedings: Influence of prostaglandin E1, indomethacin, calcium and potassium on the action of nociceptive substances.
Naunyn Schmiedebergs Arch Pharmacol. 1974;282(Suppl):suppl 282:R42.
3
Potentiation of bradykinin-induced nociceptive response by arachidonate metabolites in dogs.花生四烯酸代谢产物对犬缓激肽诱导的伤害性反应的增强作用
Eur J Pharmacol. 1986 Dec 2;132(1):47-52. doi: 10.1016/0014-2999(86)90007-5.
4
[Prostaglandin liberation by bradykinin in the rat].[缓激肽在大鼠体内释放前列腺素]
C R Seances Soc Biol Fil. 1974;168(2-3):375-8.
5
Role of prostaglandins in ventricular reflexes.前列腺素在心室反射中的作用。
Adv Prostaglandin Thromboxane Res. 1980;7:601-8.
6
An excitatory nociceptive cardiac reflex elicited by bradykinin and potentiated by prostaglandins and myocardial ischaemia.由缓激肽引发、前列腺素和心肌缺血增强的一种兴奋性伤害性心脏反射。
Cardiovasc Res. 1976 May;10(3):314-27. doi: 10.1093/cvr/10.3.314.
7
[Effects of prostaglandin E2 and sodium hyaluronate on bradykinin induced knee joint pain in rat].[前列腺素E2和透明质酸钠对缓激肽诱导的大鼠膝关节疼痛的影响]
Nihon Seikeigeka Gakkai Zasshi. 1995 Sep;69(9):735-43.
8
Inhibition of prostaglandin biosynthesis as the mechanism of analgesia of aspirin-like drugs in the dog knee joint.抑制前列腺素生物合成作为阿司匹林类药物对犬膝关节镇痛作用的机制。
Eur J Pharmacol. 1975 Apr;31(2):250-60. doi: 10.1016/0014-2999(75)90047-3.
9
The effect of indomethacin and enalapril on the cutaneous response to bradykinin.吲哚美辛和依那普利对皮肤对缓激肽反应的影响。
Br J Clin Pharmacol. 1989 Jun;27(6):870-2. doi: 10.1111/j.1365-2125.1989.tb03452.x.
10
[The hypotensive action of bradykinin in the guinea pig and cat].[缓激肽在豚鼠和猫体内的降压作用]
C R Seances Soc Biol Fil. 1976 Oct;170(3):693-7.

引用本文的文献

1
Increased susceptibility of annexin-A1 null mice to nociceptive pain is indicative of a spinal antinociceptive action of annexin-A1.膜联蛋白A1基因敲除小鼠对伤害性疼痛的易感性增加,表明膜联蛋白A1具有脊髓抗伤害感受作用。
Br J Pharmacol. 2008 Jul;154(5):1135-42. doi: 10.1038/bjp.2008.166. Epub 2008 May 12.
2
Pro-inflammatory properties of the kallikrein-kinin system: potential for new drug therapy.激肽释放酶-激肽系统的促炎特性:新药物治疗的潜力。
Inflammopharmacology. 1998;6(4):289-96. doi: 10.1007/s10787-998-0013-5.
3
Interaction of prostaglandins and indomethacin with algesic substances.前列腺素和吲哚美辛与致痛物质的相互作用。
Naunyn Schmiedebergs Arch Pharmacol. 1974;285(4):301-13. doi: 10.1007/BF00501460.
4
The effect of hyaluronan on interleukin-1 alpha-induced prostaglandin E2 production in human osteoarthritic synovial cells.透明质酸对人骨关节炎滑膜细胞中白细胞介素-1α诱导的前列腺素E2产生的影响。
Agents Actions. 1992 Sep;37(1-2):155-6. doi: 10.1007/BF01987905.
5
On the ability of prostaglandin E1, and arachidonic acid to modulate experimentally induced oedema in the rat paw.关于前列腺素E1和花生四烯酸调节大鼠爪实验性诱导水肿的能力。
Br J Pharmacol. 1975 Sep;55(1):51-6. doi: 10.1111/j.1476-5381.1975.tb07609.x.
6
Release of prostaglandins and incapacitation after injection of endotoxin in the knee joint of the dog.狗膝关节注射内毒素后前列腺素的释放与失能
Br J Pharmacol. 1975 Mar;53(3):465P.
7
Mechanism and modification of bradykinin-induced coronary vasodilation.缓激肽诱导冠状动脉舒张的机制及调节
Proc Natl Acad Sci U S A. 1975 Jun;72(6):2060-3. doi: 10.1073/pnas.72.6.2060.
8
Release of prostaglandins by bradykinin as an intrinsic mechanism of its algesic effect.缓激肽释放前列腺素作为其致痛作用的内在机制。
Naunyn Schmiedebergs Arch Pharmacol. 1976 Jul;294(1):69-73. doi: 10.1007/BF00692786.
9
Release of prostaglandins from the isolated perfused rabbit ear by bradykinin and acetylcholine.缓激肽和乙酰胆碱从离体灌注兔耳中释放前列腺素。
Agents Actions. 1976 Sep;6(5):642-5. doi: 10.1007/BF01971584.
10
Influence of changed calcium and potassium concentration on the algesic effect of bradykinin and acetylcholine.钙和钾浓度变化对缓激肽和乙酰胆碱致痛作用的影响。
Naunyn Schmiedebergs Arch Pharmacol. 1977 Oct;299(3):289-94. doi: 10.1007/BF00500323.

本文引用的文献

1
Indomethacin and aspirin abolish prostaglandin release from the spleen.吲哚美辛和阿司匹林可抑制前列腺素从脾脏释放。
Nat New Biol. 1971 Jun 23;231(25):237-9. doi: 10.1038/newbio231237a0.
2
Aspirin selectively inhibits prostaglandin production in human platelets.阿司匹林可选择性抑制人体血小板中前列腺素的生成。
Nat New Biol. 1971 Jun 23;231(25):235-7. doi: 10.1038/newbio231235a0.
3
Inhibition of prostaglandin synthesis as a mechanism of action for aspirin-like drugs.抑制前列腺素合成作为阿司匹林类药物的作用机制。
Nat New Biol. 1971 Jun 23;231(25):232-5. doi: 10.1038/newbio231232a0.
4
Prostaglandins, aspirin-like drugs and analgesia.前列腺素、阿司匹林类药物与镇痛
Nat New Biol. 1972 Dec 13;240(102):200-3. doi: 10.1038/newbio240200a0.
5
Further experiments to establish that the analgesic action of aspirin-like drugs depends on the inhibition of prostaglandin biosynthesis.进一步的实验证实,类阿司匹林药物的镇痛作用取决于对前列腺素生物合成的抑制。
Br J Pharmacol. 1973 Mar;47(3):629P-630P.