Strozzi C, Cocco G, Padovan G C
G Ital Cardiol. 1979;9(6):635-9.
18 patients with non-ischemic heart disease have been studied with reference to the kinetocardiographic changes before and after i.v. administration of dopamine (DOP) (Revivan) or Isoproterenol (ISP) (Aleudrin). For this purpose a simultaneous registration of ECG (lead D2), Kinetocardiogram (KCG) in K25 and K45 was performed. Volume curves were considered as a normal features of KCG, curves showing systolic outward movements during the ejection phase were classified as pathologic. The reduction or disappearance of the systolic movements during the drug administration, were considered as kinetic improvement, the increase of their site was judged as kinetic worsening. During the infusion, 17 KCGs maintained a normal morphological pattern; out of 19 pathological KCGs, 4 did not show any change, and 15, 5 reached a complete normalization. It is pointed out the overlap of the precordial kinetics in the ventricular hypertrophies and in the normal subjects (kinetic improvement), and the possibility to differentiate a systolic outward movement due to synergic contraction of hypertrophied ventricle from a systolic outward movement to be reffered to a diskynetic contraction in the myocardial ischemia (kinetic worsening).
对18例非缺血性心脏病患者进行了研究，观察静脉注射多巴胺（DOP，商品名Revivan）或异丙肾上腺素（ISP，商品名Aleudrin）前后的心动力学变化。为此，同步记录心电图（D2导联）、心动力学图（KCG）在K25和K45时的情况。容积曲线被视为KCG的正常特征，在射血期显示收缩期向外运动的曲线被归类为病理性曲线。给药期间收缩期运动的减少或消失被视为动力学改善，其范围的增加被判定为动力学恶化。输液过程中，17份KCG保持正常形态模式；19份病理性KCG中，4份未显示任何变化，15份中有5份完全恢复正常。指出心室肥厚患者和正常受试者的心前区动力学存在重叠（动力学改善），以及区分肥厚心室协同收缩引起的收缩期向外运动与心肌缺血中运动障碍性收缩导致的收缩期向外运动的可能性（动力学恶化）。
