费尔蒂综合征中的粒细胞生成活性。
Granulopoietic activity in Felty's syndrome.
作者信息
Gupta R, Robinson W A, Albrecht D
出版信息
Ann Rheum Dis. 1975 Apr;34(2):156-61. doi: 10.1136/ard.34.2.156.
Abstract
The levels of urinary and serum granulopoietic factor have been determined in nine patients with Felty's syndrome and compared with similar levels in patients with rheumatoid arthritis without Felty's syndrome and patients with neutropenic disorders of other causes. These studies have shown that urinary and serum levels of colony stimulating activity (CSA) in patients with Felty's syndrome are low (mean urine CSA 10.0 and serum CSA 4.0) when compared to patients with neutropenic disorders without rheumatoid arthritis (mean urinary CSA 26.8 and serum CSA 9.4). These findings suggest that Felty's syndrome may be, in part, the result of decreased production or activity of granulopoietic factors rather than due simply to increased granulocyte destruction.
摘要
已测定9例费尔蒂综合征患者尿液和血清中的粒细胞生成因子水平，并与无费尔蒂综合征的类风湿关节炎患者及其他原因导致的中性粒细胞减少症患者的类似水平进行了比较。这些研究表明，与无类风湿关节炎的中性粒细胞减少症患者（平均尿集落刺激活性26.8，血清集落刺激活性9.4）相比，费尔蒂综合征患者尿液和血清中的集落刺激活性（CSA）水平较低（平均尿CSA 10.0，血清CSA 4.0）。这些发现提示，费尔蒂综合征可能部分是粒细胞生成因子产生减少或活性降低的结果，而不仅仅是粒细胞破坏增加所致。
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