急性感染所致嗜酸性粒细胞减少症的实验观察
Experimental observations on the eosinopenia induced by acute infection.
作者信息
Morgan J E, Beeson P B
出版信息
Br J Exp Pathol. 1971 Apr;52(2):214-20.
Abstract
The effect of trichinosis on the number of eosinophils in the peripheral circulation and in the marrow has been determined in mice by accurate techniques. There is a sharp rise in number of eosinophils in the bone marrow and in the peripheral blood beginning about the 10th day, which persists for some weeks. The correlation between peripheral blood and evidence of bone marrow production is remarkably close. When acute pyelonephritis is induced during the height of eosinophilia due to trichinosis these cells rapidly disappear from peripheral blood and bone marrow, so that by the end of 24-48 hr the counts are in or near the normal range.
摘要
通过精确技术已在小鼠中确定旋毛虫病对其外周循环和骨髓中嗜酸性粒细胞数量的影响。从大约第10天开始，骨髓和外周血中的嗜酸性粒细胞数量急剧上升，并持续数周。外周血与骨髓产生证据之间的相关性非常密切。当在旋毛虫病引起的嗜酸性粒细胞增多高峰期诱发急性肾盂肾炎时，这些细胞会迅速从外周血和骨髓中消失，以至于在24至48小时结束时计数处于或接近正常范围。
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本文引用的文献
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Experimental pyelonephritis. I. Effect of ureteral ligation on the course of bacterial infection in the kidney of the rat.实验性肾盂肾炎。I. 输尿管结扎对大鼠肾脏细菌感染病程的影响。
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Mechanism of eosinophilia. II. Role of the lymphocyte.嗜酸性粒细胞增多的机制。II. 淋巴细胞的作用。
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Mechanism of eosinophilia. I. Factors affecting the eosinophil response of rats to Trichinella spiralis.嗜酸性粒细胞增多的机制。I. 影响大鼠对旋毛虫嗜酸性粒细胞反应的因素。
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