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"Tertiary" hyperparathyroidism induced by osteomalacia resulting from phosphorus depletion.

作者信息

Ludwig G D, Kyle C G, de Blanco M

出版信息

Am J Med. 1967 Jul;43(1):136-40. doi: 10.1016/0002-9343(67)90155-6.

DOI:10.1016/0002-9343(67)90155-6
PMID:4951413
Abstract
摘要

相似文献

1
"Tertiary" hyperparathyroidism induced by osteomalacia resulting from phosphorus depletion.由磷缺乏导致骨软化症引起的“三发性”甲状旁腺功能亢进。
Am J Med. 1967 Jul;43(1):136-40. doi: 10.1016/0002-9343(67)90155-6.
2
[Primary hyperparathyroidism with osteomalacia caused by parathyroid adenoma: analysis of 10 cases].
Zhonghua Nei Ke Za Zhi. 1986 Feb;25(2):69-72, 125.
3
Primary hyperparathyroidism associated with nutritional osteomalacia.
Am J Med. 1969 Apr;46(4):640-4. doi: 10.1016/0002-9343(69)90083-7.
4
Association between prolonged dietary vitamin D deficiency and autonomous hyperparathyroidism.长期膳食维生素D缺乏与自主性甲状旁腺功能亢进之间的关联。
BMJ. 1989 Jul 22;299(6693):236-7. doi: 10.1136/bmj.299.6693.236.
5
[Tertiary hyperparathyroidism revealing celiac disease in adults].[成人腹腔疾病并发的三发性甲状旁腺功能亢进]
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6
Tumor-related osteomalacia followed after treatment by hyperparathyroidism.
Rev Rhum Engl Ed. 1999 Jan;66(1):53-7.
7
[Osteomalacia symptoms in primary hyperparathyroidism].[原发性甲状旁腺功能亢进症中的骨软化症状]
Rev Rhum Mal Osteoartic. 1988 May;55(7):505-10.
8
Letter: Masked primary (or tertiary) hyperparathyroidism.信函：隐匿性原发性（或三发性）甲状旁腺功能亢进症。
Lancet. 1975 Sep 13;2(7933):503. doi: 10.1016/s0140-6736(75)90578-4.
9
Prolonged neonatal parathyroid suppression. A sequel to asymptomatic maternal hyperparathyroidism.新生儿甲状旁腺长期受抑制。无症状母亲甲状旁腺功能亢进的后遗症。
Arch Surg. 1973 May;106(5):722-4. doi: 10.1001/archsurg.1973.01350170082022.
10
Hyperparathyroidism related to an ectopic parathyroid adenoma.与异位甲状旁腺腺瘤相关的甲状旁腺功能亢进症。
Pan Afr Med J. 2014 Oct 8;19:135. doi: 10.11604/pamj.2014.19.135.4931. eCollection 2014.

引用本文的文献

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Specific genetic aberrations of parathyroid in Chinese patients with tertiary hyperparathyroidism using whole-exome sequencing.使用全外显子组测序分析中国三发性甲状旁腺功能亢进症患者甲状旁腺的特定基因突变。
Front Endocrinol (Lausanne). 2023 Oct 3;14:1221060. doi: 10.3389/fendo.2023.1221060. eCollection 2023.
2
Vitamin D-Mediated Hypercalcemia: Mechanisms, Diagnosis, and Treatment.维生素D介导的高钙血症：机制、诊断与治疗
Endocr Rev. 2016 Oct;37(5):521-547. doi: 10.1210/er.2016-1070. Epub 2016 Sep 2.
3
Antacid-induced osteomalacia.抗酸剂所致骨软化症
Clin Rheumatol. 1996 Jan;15(1):75-80. doi: 10.1007/BF02231691.
4
Tertiary hyperparathyroidism.三发性甲状旁腺功能亢进症。
Br Med J. 1968 Aug 17;3(5615):395-9. doi: 10.1136/bmj.3.5615.395.
5
Hypocalcaemic primary hyperparathyroidism.低钙血症性原发性甲状旁腺功能亢进症
Br Med J. 1970 Jan 24;1(5690):208-11. doi: 10.1136/bmj.1.5690.208.
6
Antacid induction of phosphate depletion syndrome in renal failure.肾衰竭中抗酸剂诱发的磷酸盐耗竭综合征
West J Med. 1974 Feb;120(2):157-60.
7
Increased prothrombin time and metabolic changes with high serum aluminum levels following long-term exposure to Bayer-process alumina.长期接触拜耳法氧化铝后，凝血酶原时间延长以及血清铝水平升高伴随代谢变化。
Can Med Assoc J. 1971 Dec 18;105(12):1297-9 passim.
8
Antacid and sucralfate-induced hypophosphatemic osteomalacia: a case report and review of the literature.抗酸剂和硫糖铝所致低磷性骨软化症：一例报告及文献复习
Calcif Tissue Int. 1990 Nov;47(5):291-5. doi: 10.1007/BF02555911.
9
[The analyses of aluminum in serum and urine for monitoring of exposed persons (author's transl)].[血清和尿液中铝的分析用于监测接触者（作者译）]
Int Arch Occup Environ Health. 1976 Oct 21;38(1):1-17. doi: 10.1007/BF00378316.
10
The pathophysiology and clinical aspects of hypercalcemic disorders.高钙血症疾病的病理生理学与临床方面
West J Med. 1978 Oct;129(4):278-320.