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Two anticomplementary factors in cobra venom: hemolysis of guinea pig erythrocytes by one of them.

作者信息

Ballow M, Cochrane C G

出版信息

J Immunol. 1969 Nov;103(5):944-52.

PMID:4981797
Abstract
摘要

相似文献

1
Two anticomplementary factors in cobra venom: hemolysis of guinea pig erythrocytes by one of them.眼镜蛇毒中的两种抗补体因子：其中一种可使豚鼠红细胞发生溶血。
J Immunol. 1969 Nov;103(5):944-52.
2
Isolation of the anticomplementary protein from cobra venom and its mode of action on C3.眼镜蛇毒中抗补体蛋白的分离及其对C3的作用方式。
J Immunol. 1971 Dec;107(6):1666-72.
3
Bypass-activation of the complement system starting with C3. I. Generation and function of an enzyme from a factor of guinea-pig serum and cobra venom.补体系统从C3开始的旁路激活。I. 由豚鼠血清因子和眼镜蛇毒产生的一种酶及其功能。
Immunology. 1972 Sep;23(3):267-81.
4
Serum proteins involved in decay and regeneration of cobra venom factor-dependent complement activation.参与眼镜蛇毒因子依赖性补体激活的衰变和再生过程的血清蛋白。
J Immunol. 1973 Dec;111(6):1730-6.
5
Depletion of plasma complement in vivo by a protein of cobra venom: its effect on various immunologic reactions.眼镜蛇毒蛋白在体内对血浆补体的消耗：其对各种免疫反应的影响。
J Immunol. 1970 Jul;105(1):55-69.
6
Blocking of the hemolytic expression of the classical complement sequence by products of complement activation via the alternate pathway. Materials responsible for the blocking phenomenon and their proposed mechanism of action.通过替代途径激活补体产生的产物对经典补体序列溶血表达的阻断。负责阻断现象的物质及其提出的作用机制。
J Immunol. 1973 Feb;110(2):390-400.
7
Production of antibodies monospecifically reactive with the third component of guinea pig complement.产生与豚鼠补体第三成分单特异性反应的抗体。
Int Arch Allergy Appl Immunol. 1970;37(2):124-42. doi: 10.1159/000230227.
8
Immune hemolysis and a basic peptide from cobra venom.免疫溶血与眼镜蛇毒中的一种碱性肽。
J Immunol. 1974 Nov;113(5):1647-9.
9
Studies on eosinophil leucocyte migration. II. Factors specifically chemotactic for eosinophils and neutrophils generated from guinea-pig serum by antigen-antibody complexes.嗜酸性白细胞迁移的研究。II. 抗原抗体复合物从豚鼠血清中产生的对嗜酸性粒细胞和中性粒细胞具有特异性趋化作用的因子。
Clin Exp Immunol. 1970 Nov;7(5):723-37.
10
Formation and function of a complement-activating enzyme generated from factors of guinea pig serum and cobra venom.
Eur J Immunol. 1971 Aug;1(4):309-11. doi: 10.1002/eji.1830010421.

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The properdin pathway: an "alternative activation pathway" or a "critical amplification loop" for C3 and C5 activation?备解素途径：C3 和 C5 激活的“替代激活途径”还是“关键扩增环”？
Semin Immunopathol. 2018 Jan;40(1):15-35. doi: 10.1007/s00281-017-0661-x. Epub 2017 Nov 22.
2
Prevention of the localized and generalized Shwartzman reactions by an anticomplementary agent, cobra venom factor.用一种抗补体剂——眼镜蛇毒因子预防局部性和全身性施瓦茨曼反应。
J Exp Med. 1971 Sep 1;134(3 Pt 1):642-55. doi: 10.1084/jem.134.3.642.
3
Effects of two types of cobra venom factor on porcine complement activation and pulmonary artery pressure.
两种眼镜蛇毒因子对猪补体激活及肺动脉压的影响。
Clin Exp Immunol. 1989 Nov;78(2):299-306.
4
The glomerular mesangium. II. Studies of macromolecular uptake in nephrotoxic nephritis in rats.肾小球系膜。II. 大鼠肾毒性肾炎中大分子摄取的研究。
J Clin Invest. 1974 Feb;53(2):431-9. doi: 10.1172/JCI107577.
5
Complement depletion facilitates the infection of multiple brain tumors by an intravascular, replication-conditional herpes simplex virus mutant.补体耗竭促进血管内复制条件性单纯疱疹病毒突变体对多种脑肿瘤的感染。
J Virol. 2000 May;74(10):4765-75. doi: 10.1128/jvi.74.10.4765-4775.2000.
6
Mediators of microvascular injury in dermal burn wounds.皮肤烧伤创面微血管损伤的介质
Inflammation. 1998 Dec;22(6):619-29. doi: 10.1023/a:1022366514847.
7
Differing patterns of P-selectin expression in lung injury.肺损伤中P-选择素表达的不同模式。
Am J Pathol. 1998 Oct;153(4):1113-22. doi: 10.1016/S0002-9440(10)65655-6.
8
P-selectin requirement for neutrophil accumulation and injury in the direct passive Arthus reaction.直接被动Arthus反应中中性粒细胞聚集和损伤对P-选择素的需求。
Clin Exp Immunol. 1998 May;112(2):281-6. doi: 10.1046/j.1365-2249.1998.00584.x.
9
Neutrophil chemotactic activity and C5a following systemic activation of complement in rats.大鼠补体系统全身激活后的中性粒细胞趋化活性与C5a
Inflammation. 1997 Jun;21(3):325-33. doi: 10.1023/a:1027302017117.
10
Increased migration of neutrophils to granulocyte-colony stimulating factor in rat carrageenin-induced pleurisy: roles of complement, bradykinin, and inducible cyclooxygenase-2.在大鼠角叉菜胶诱导的胸膜炎中，中性粒细胞向粒细胞集落刺激因子的迁移增加：补体、缓激肽和诱导型环氧化酶-2的作用
Inflamm Res. 1996 Jul;45(7):335-46. doi: 10.1007/BF02252946.