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Effects of coronary blood flow and perfusion pressure on left ventricular contractility in dogs.

作者信息

Abel R M, Reis R L

出版信息

Circ Res. 1970 Dec;27(6):961-71. doi: 10.1161/01.res.27.6.961.

DOI:10.1161/01.res.27.6.961
PMID:4992167
Abstract
摘要

相似文献

1
Effects of coronary blood flow and perfusion pressure on left ventricular contractility in dogs.冠状动脉血流量和灌注压对犬左心室收缩性的影响。
Circ Res. 1970 Dec;27(6):961-71. doi: 10.1161/01.res.27.6.961.
2
Effects of catecholamines, exercise, and nitroglycerin on the normal and ischemic myocardium in conscious dogs.儿茶酚胺、运动及硝酸甘油对清醒犬正常及缺血心肌的影响。
J Clin Invest. 1974 Sep;54(3):563-75. doi: 10.1172/JCI107793.
3
Direct and reflex effects of nitroglycerin on coronary and left ventricular dynamics in conscious dogs.硝酸甘油对清醒犬冠状动脉和左心室动力学的直接及反射作用
J Clin Invest. 1972 Nov;51(11):2872-82. doi: 10.1172/JCI107110.
4
[Physiology of the coronary circulation as a basis for evaluation of coronary dilators].[作为评估冠状动脉扩张剂基础的冠状动脉循环生理学]
Naunyn Schmiedebergs Arch Exp Pathol Pharmakol. 1969;263(1):127-44.
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Myocardial oxygen consumption of blood-perfused, isolated, supported, rat heart.血液灌注的离体支持大鼠心脏的心肌耗氧量。
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6
[Physiology of the coronary circulation as a basis for evaluating coronary dilators].[作为评估冠状动脉扩张剂基础的冠状动脉循环生理学]
Kardiologiia. 1972 Jan;12(1):22-31.
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The importance of the perfusion pressure in the coronary arteries for the contractility and the oxygen consumption of the heart.冠状动脉灌注压对心脏收缩性和氧消耗的重要性。
Pflugers Arch Gesamte Physiol Menschen Tiere. 1968;299(4):339-56. doi: 10.1007/BF00602910.
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Comparative profile of L 3428 and other antianginal agents on cardiac hemodynamics.L 3428与其他抗心绞痛药物对心脏血流动力学的比较概况。
J Pharmacol Exp Ther. 1971 Feb;176(2):328-38.
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[Action of Persantin and nitroglycerin on systemic and coronary hemodynamics in the dog. Preliminary note].[潘生丁和硝酸甘油对犬全身及冠状动脉血流动力学的作用。初步报告]
Minerva Cardioangiol. 1970 Oct;18(10):589-93.
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Coronary perfusion pressure and left ventricle function.冠状动脉灌注压与左心室功能。
Pflugers Arch. 1969;305(1):76-95. doi: 10.1007/BF00586397.

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心肌血流储备和静息收缩功能调节接受铷-82正电子发射断层扫描患者的收缩储备影响。
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Non-linearity of end-systolic pressure-volume relation in afterload increases is caused by an overlay of shortening deactivation and the Frank-Starling mechanism.后负荷增加时,收缩失活和 Frank-Starling 机制的叠加导致收缩末期压力-容积关系的非线性。
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