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Inhibition of mouse skin tumorigenesis by actinomycin D.

作者信息

Gelboin H V, Klein M, Bates R R

出版信息

Proc Natl Acad Sci U S A. 1965 Jun;53(6):1353-60. doi: 10.1073/pnas.53.6.1353.

DOI:10.1073/pnas.53.6.1353
PMID:5217637
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC219855/
Abstract
摘要

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On the biochemical mechanism of tumorigenesis in mouse skin. II. Early effects on the biosynthesis of nucleic acids induced by initiating doses of DMBA and by promoting doses of phorbol-12.13-diester TPA.关于小鼠皮肤肿瘤发生的生化机制。II. 起始剂量的二甲基苯蒽（DMBA）和促癌剂量的佛波醇-12,13-二酯（TPA）对核酸生物合成的早期影响。
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Inhibition by actinomycin D of DNA synthesis and skin tumorigenesis induced by 7,12-dimethylbenz(a)anthracene.放线菌素D对7,12-二甲基苯并(a)蒽诱导的DNA合成及皮肤肿瘤发生的抑制作用。
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[On the biochemical mechanism of tumorgenesis in the mouse skin. I. Distribution and metabolism of intragastrically administered 9,10-dimethyl-(1,2)-benzanthracene in the mouse].[关于小鼠皮肤肿瘤发生的生化机制。I. 经胃给予小鼠的9,10-二甲基-（1,2）-苯并蒽的分布与代谢]
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A test for mutation theory of cancer: carcinogenesis by misrepair of DNA damaged by 4-nitroquinoline 1-oxide.癌症突变理论的一项测试：4-硝基喹啉 1-氧化物损伤 DNA 的错配修复导致致癌作用。
Br J Cancer. 1977 May;35(5):595-601. doi: 10.1038/bjc.1977.93.

本文引用的文献

1
Uptake of radioactive alanine in vitro into the proteins of rat liver fractions.体外放射性丙氨酸在大鼠肝脏各组分蛋白质中的摄取。
J Biol Chem. 1952 Apr;195(2):549-65.
2
Teleonomic mechanisms in cellular metabolism, growth, and differentiation.细胞代谢、生长和分化中的目的性机制。
Cold Spring Harb Symp Quant Biol. 1961;26:389-401. doi: 10.1101/sqb.1961.026.01.048.
3
RECOGNITION OF ALTERED PATTERNS OF MESSENGER RNA SYNTHESIS IN A MOUSE HEPATOMA.小鼠肝癌中信使核糖核酸合成模式改变的识别
Cancer Res. 1964 Oct;24:1604-9.
4
STUDIES ON THE MECHANISM OF METHYLCHOLANTHRENE INDUCTION OF ENZYME ACTIVITIES. II. STIMULATION OF MICROSOMAL AND RIBOSOMAL AMINO ACID INCORPORATION: THE EFFECTS OF POLYURIDYLIC ACID AND ACTINOMYCIN-D.甲基胆蒽诱导酶活性的机制研究。II. 对微粒体和核糖体氨基酸掺入的刺激作用：多聚尿苷酸和放线菌素-D的影响。
Biochim Biophys Acta. 1964 Sep 11;91:130-44.
5
A PARADOXICAL EFFECT OF ACTINOMYCIN D: THE MECHANISM OF REGULATION OF ENZYME SYNTHESIS BY HYDROCORTISONE.放线菌素D的一种矛盾效应：氢化可的松对酶合成的调节机制
Proc Natl Acad Sci U S A. 1964 Oct;52(4):1121-9. doi: 10.1073/pnas.52.4.1121.
6
INHIBITION BY A CARCINOGENIC HYDROCARBON OF INCORPORATION OF TRITIATED CYTIDINE INTO MOUSE EPIDERMAL CELLS.一种致癌碳氢化合物对氚标记胞苷掺入小鼠表皮细胞的抑制作用。
Nature. 1964 Aug 1;203:521-3. doi: 10.1038/203521a0.
7
EFFECTS OF POLYCYCLIC AROMATIC CARCINOGENS ON VIRAL REPLICATION: SIMILARITY TO ACTINOMYCIN D.多环芳烃致癌物对病毒复制的影响：与放线菌素D的相似性
Science. 1964 Oct 30;146(3644):650-1. doi: 10.1126/science.146.3644.650.
8
EVIDENCE FOR THE BINDING OF POLYNUCLEAR AROMATIC HYDROCARBONS TO THE NUCLEIC ACIDS OF MOUSE SKIN: RELATION BETWEEN CARCINOGENIC POWER OF HYDROCARBONS AND THEIR BINDING TO DEOXYRIBONUCLEIC ACID.多环芳烃与小鼠皮肤核酸结合的证据：烃类致癌能力与其与脱氧核糖核酸结合之间的关系。
Nature. 1964 May 23;202:781-4. doi: 10.1038/202781a0.
9
SKIN TUMORIGENESIS BY 7,12-DIMETHYLBENZ(A)ANTHRACENE: INHIBITION BY ACTINOMYCIN D.7,12-二甲基苯并(a)蒽诱发皮肤肿瘤：放线菌素D的抑制作用
Science. 1964 Sep 18;145(3638):1321-2. doi: 10.1126/science.145.3638.1321.
10
POLYCYCLIC HYDROCARBONS.多环烃类
Br Med Bull. 1964 May;20:121-6. doi: 10.1093/oxfordjournals.bmb.a070304.