Suppression of the shunt pathway in primary gout by azathioprine.
作者信息
Sorensen L B
出版信息
Proc Natl Acad Sci U S A. 1966 Mar;55(3):571-5. doi: 10.1073/pnas.55.3.571.
Abstract
摘要
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Suppression of the shunt pathway in primary gout by azathioprine.硫唑嘌呤对原发性痛风分流途径的抑制作用。
Proc Natl Acad Sci U S A. 1966 Mar;55(3):571-5. doi: 10.1073/pnas.55.3.571.
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Suppression of glycine-15N incorporation into urinary uric acid by adenine-8-13C in normal and gouty subjects.在正常人和痛风患者中，腺嘌呤 -8-13C 对甘氨酸 -15N 掺入尿尿酸的抑制作用。
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Uric acid production in normal and gouty subjects, determined by N15 labeled glycine.通过N15标记甘氨酸测定正常人和痛风患者的尿酸生成。
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A specific enzyme defect in gout associated with overproduction of uric acid.痛风中与尿酸生成过多相关的一种特定酶缺陷。
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引用本文的文献
1
The effects of azathioprine (imuran) on purine synthesis in clinical disorders of purine metabolism.巯嘌呤（依木兰）对嘌呤代谢临床紊乱中嘌呤合成的影响。
J Clin Invest. 1967 Sep;46(9):1518-29. doi: 10.1172/JCI105643.
2
Current concepts of hyperuricemia and gout.高尿酸血症与痛风的当前概念。
Calif Med. 1969 Mar;110(3):231-44.
3
Mechanism of excessive purine biosynthesis in hypoxanthine-guanine phosphoribosyltransferase deficiency.次黄嘌呤-鸟嘌呤磷酸核糖转移酶缺乏症中嘌呤生物合成过多的机制。
J Clin Invest. 1970 May;49(5):968-78. doi: 10.1172/JCI106316.
本文引用的文献
1
THE RENAL EXCRETION OF URIC ACID IN GOUT.痛风患者尿酸的肾脏排泄
J Clin Invest. 1962 May;41(5):1094-8. doi: 10.1172/JCI104560.
2
3
AN ABNORMALITY OF GLUTAMINE METABOLISM IN PRIMARY GOUT.原发性痛风中谷氨酰胺代谢异常
Am J Med. 1963 Dec;35:820-31. doi: 10.1016/0002-9343(63)90244-4.
4
The pathogenesis of gout.痛风的发病机制。
Arch Intern Med. 1962 Apr;109:379-90. doi: 10.1001/archinte.1962.03620160005002.
5
The pathogenesis of gout.痛风的发病机制。
Med Clin North Am. 1961 Sep;45:1241-58. doi: 10.1016/s0025-7125(16)33832-9.
6
Enzymatic determination of uric acid; with detailed directions.尿酸的酶法测定；详细说明。
Scand J Clin Lab Invest. 1953;5(3):273-80. doi: 10.3109/00365515309094197.
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