Adams P, Chalmers T M, Hill L F, Truscott B M
Br Med J. 1970 Dec 5;4(5735):582-5. doi: 10.1136/bmj.4.5735.582.
With provocative tests of parathyroid activity based on phosphate deprivation and the administration of chlorothiazide 8 out of 19 patients with idiopathic hypercalciuria developed hypercalcaemia. Five of the eight underwent exploration of the neck and four of them had parathyroid adenomas; the fifth patient had "normal" glands but responded satisfactorily to subtotal parathyroidectomy. One patient who failed to develop hypercalcaemia had primary chief cell hyperplasia of the parathyroids.
基于磷缺乏和给予氯噻嗪进行甲状旁腺活性激发试验，19例特发性高钙尿症患者中有8例出现高钙血症。8例中的5例接受了颈部探查，其中4例有甲状旁腺腺瘤；第5例患者甲状旁腺“正常”，但次全甲状旁腺切除术后反应良好。1例未出现高钙血症的患者患有甲状旁腺原发性主细胞增生。
