Dessì M A, Dore M, Montaldo C, Sanna A
Boll Soc Ital Biol Sper. 1979 Feb 28;55(4):361-5.
The rats were pretreated with cadmium acetate (20 mg/Kg.ip) and subsequently challenged with a dose of bromotrichloromethane (2,6 mmoles/Kg.po), the haloalkane produced no change on liver polyribosomes, as evidenced by disaggregation of sedimentation profile. Cadmium acetate failed to protect against CBrCl3-induced lipid peroxidation, as measured by malonaldehyde production of liver homogenate.
大鼠先用醋酸镉（20毫克/千克，腹腔注射）进行预处理，随后用一定剂量的溴三氯甲烷（2.6毫摩尔/千克，口服）进行激发，卤代烷对肝脏多核糖体没有产生变化，沉降图谱的解聚证明了这一点。通过肝脏匀浆丙二醛生成量测定，醋酸镉未能预防三氯溴甲烷诱导的脂质过氧化。
