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Incorporation of oleic acid into lipid by foam cells in human atherosclerotic lesions.

作者信息

Wahlqvist M L, Day A J, Tume R K

出版信息

Circ Res. 1969 Jan;24(1):123-30. doi: 10.1161/01.res.24.1.123.

DOI:10.1161/01.res.24.1.123
PMID:5763735
Abstract
摘要

相似文献

1
Incorporation of oleic acid into lipid by foam cells in human atherosclerotic lesions.
Circ Res. 1969 Jan;24(1):123-30. doi: 10.1161/01.res.24.1.123.
2
In vitro incorporation of 14C-labelled oleic acid into combined lipid by foam cells from rabbit atheromatous lesions.兔动脉粥样硬化病变泡沫细胞将¹⁴C标记的油酸体外掺入复合脂质中。
J Atheroscler Res. 1969 Mar-Apr;9(2):141-9. doi: 10.1016/s0368-1319(69)80049-9.
3
Incorporation of (14C)oleic acid into lipid by foam cells and by other fractions separated from rabbit atherosclerotic lesions.泡沫细胞以及从兔动脉粥样硬化病变中分离出的其他组分将（14C）油酸掺入脂质的过程。
Atherosclerosis. 1970 Mar-Apr;11(2):291-9. doi: 10.1016/0021-9150(70)90067-5.
4
Uptake and metabolism of 14C-labeled oleic acid by atherosclerotic lesions in rabbit aorta.兔主动脉粥样硬化病变对14C标记油酸的摄取与代谢
Circ Res. 1968 Dec;23(6):779-83. doi: 10.1161/01.res.23.6.779.
5
Lipid metabolism of the arterial wall in thromboangiitis obliterans (Buerger's disease).
Virchows Arch A Pathol Anat Histol. 1975 Dec 31;369(2):123-30. doi: 10.1007/BF00433238.
6
Ultrastructure, fatty acid content, and metabolic activity of foam cells and other fractions separated from rabbit atherosclerotic lesions.从兔动脉粥样硬化病变中分离出的泡沫细胞及其他组分的超微结构、脂肪酸含量和代谢活性
Exp Mol Pathol. 1971 Oct;15(2):157-69. doi: 10.1016/0014-4800(71)90096-7.
7
Localization by autoradiography of phospholipid synthesis in rabbit atherosclerotic aorta.兔动脉粥样硬化主动脉中磷脂合成的放射自显影定位
Exp Mol Pathol. 1969 Dec;11(3):263-74. doi: 10.1016/0014-4800(69)90014-8.
8
Effects of regression of atherosclerotic lesions on the content and esterification of cholesterol by cell-free preparations of pigeon aorta.鸽主动脉无细胞制剂中动脉粥样硬化病变消退对胆固醇含量及酯化的影响。
Circ Res. 1972 Nov;31(5):664-71. doi: 10.1161/01.res.31.5.664.
9
Phospholipid synthesis by foam cells in human.
Exp Mol Pathol. 1969 Dec;11(3):275-84. doi: 10.1016/0014-4800(69)90015-x.
10
[Proceedings: Incorporation of 1-14C-oleic acid in subcellular fractions or normal and atherosclerotic human femoral arteries in vitro].
MMW Munch Med Wochenschr. 1974 May 31;116(22):1111-2.

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Imagining a habitable planet through food and health.通过食物和健康来想象一个宜居的星球。
Eur J Clin Nutr. 2021 Feb;75(2):219-229. doi: 10.1038/s41430-020-0672-x. Epub 2020 Jun 17.
2
Lipid droplets in atherosclerotic fatty streaks of human aorta.人主动脉动脉粥样硬化脂肪条纹中的脂滴。
J Clin Invest. 1970 Aug;49(8):1479-88. doi: 10.1172/JCI106365.
3
Phospholipases in arterial tissue. IV. The role of phosphatide acyl hydrolase, lysophosphatide acyl hydrolase, and sphingomyelin choline phosphohydrolase in the regulation of phospholipid composition in the normal human aorta with age.
动脉组织中的磷脂酶。IV. 磷脂酰基水解酶、溶血磷脂酰基水解酶和鞘磷脂胆碱磷酸水解酶在正常人体主动脉磷脂组成随年龄调节中的作用。
J Clin Invest. 1969 Dec;48(12):2320-9. doi: 10.1172/JCI106198.
4
Lipid metabolism by rabbit aortic intimal and medial cells in tissue culture.
Virchows Arch A Pathol Anat Histol. 1974 Jan 30;362(2):89-105. doi: 10.1007/BF00432388.
5
The effect of insulin on the incorporation of sodium (1-14C)-acetate into the lipids of the rat aorta.
Diabetologia. 1972 Aug;8(4):305-6. doi: 10.1007/BF01225577.
6
The turnover of cholesterol in human atherosclerotic arteries.人类动脉粥样硬化动脉中胆固醇的周转
J Clin Invest. 1974 Aug;54(2):366-77. doi: 10.1172/JCI107772.
7
Lipoprotein degradation and cholesterol esterification in primary cell cultures of rabbit atherosclerotic lesions.兔动脉粥样硬化病变原代细胞培养中的脂蛋白降解与胆固醇酯化
Am J Pathol. 1990 Aug;137(2):457-65.
8
Lipid metabolism of the arterial wall in thromboangiitis obliterans (Buerger's disease).
Virchows Arch A Pathol Anat Histol. 1975 Dec 31;369(2):123-30. doi: 10.1007/BF00433238.
9
The lipid metabolism of the arterial wall and its abnormalities in diabetes.动脉壁的脂质代谢及其在糖尿病中的异常。
Acta Diabetol Lat. 1976 May-Aug;13(3-4):87-92. doi: 10.1007/BF02581251.
10
Reversible accumulation of cholesteryl esters in macrophages incubated with acetylated lipoproteins.与乙酰化脂蛋白一起孵育的巨噬细胞中胆固醇酯的可逆性蓄积。
J Cell Biol. 1979 Sep;82(3):597-613. doi: 10.1083/jcb.82.3.597.