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本文引用的文献

1
Toxic effects of oxygen at high pressure on the metabolism of d-glucose by dispersions of rat brain.高压氧对大鼠脑匀浆中d-葡萄糖代谢的毒性作用。
Biochem J. 1963 Jul;88(1):31-45. doi: 10.1042/bj0880031.
2
Toxic effects of oxygen and of hydrogen peroxide on brain metabolism.氧气和过氧化氢对脑代谢的毒性作用。
Biochem J. 1946;40(1):139-44. doi: 10.1042/bj0400139.
3
Pulmonary damage due to high pressure oxygen breathing in rats.大鼠高压氧呼吸所致的肺损伤
Aust J Exp Biol Med Sci. 1962 Aug;40:309-14. doi: 10.1038/icb.1962.34.
4
Pulmonary damage due to high pressure oxygen breathing in rats. 2. Changes in dehydrogenase activity of rat lung.大鼠高压氧呼吸所致的肺损伤。2. 大鼠肺组织脱氢酶活性的变化
Aust J Exp Biol Med Sci. 1962 Feb;40:51-6. doi: 10.1038/icb.1962.7.
5
THE EFFECT OF OXYGEN ON GLUTAMIC ACID DECARBOXYLASE AND GAMMA-AMINOBUTYRIC ACID-ALPHA-KETOGLUTARIC ACID TRANSAMINASE ACTIVITIES IN RAT BRAIN HOMOGENATES.氧气对大鼠脑匀浆中谷氨酸脱羧酶和γ-氨基丁酸-α-酮戊二酸转氨酶活性的影响。
Can J Physiol Pharmacol. 1964 Mar;42:277-9. doi: 10.1139/y64-032.
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MOLECULAR STRUCTURE-ACTIVITY RELATIONSHIPS OF COMPOUNDS PROTECTING RATS AGAINST OXYGEN POISONING.保护大鼠免受氧中毒的化合物的分子结构-活性关系
Can J Physiol Pharmacol. 1964 Sep;42:641-6. doi: 10.1139/y64-070.
7
BRAIN DAMAGE AND PARALYSIS IN ANIMALS EXPOSED TO HIGH PRESSURE OXYGEN--PHARMACOLOGICAL AND BIOCHEMICAL OBSERVATIONS.暴露于高压氧的动物的脑损伤和麻痹——药理学和生物化学观察
Biochem Pharmacol. 1964 Feb;13:165-82. doi: 10.1016/0006-2952(64)90134-0.
8
PULMONARY DAMAGE DUE TO HIGH PRESSURE OXYGEN BREATHING IN RATS. 4. QUANTITATIVE ANALYSIS OF SULPHYDRYL AND DISULPHIDE GROUPS IN RAT LUNGS.大鼠高压氧呼吸所致的肺损伤。4. 大鼠肺中巯基和二硫键基团的定量分析。
Aust J Exp Biol Med Sci. 1963 Oct;41:491-7. doi: 10.1038/icb.1963.38.
9
GAMMA-AMINOBUTYRIC ACID LEVELS IN THE BRAIN OF RATS EXPOSED TO OXYGEN AT HIGH PRESSURES.暴露于高压氧环境下的大鼠大脑中的γ-氨基丁酸水平
Can J Biochem Physiol. 1963 Sep;41:1907-13.
10
GAMMA-AMINOBUTYRIC ACID AND OXYGEN POISONING.γ-氨基丁酸与氧中毒
J Neurochem. 1963 Sep;10:625-33. doi: 10.1111/j.1471-4159.1963.tb08934.x.

高压氧对大鼠肝脏线粒体细胞色素稳态的影响。

Effect of high pressure oxygen on the steady state of cytochromes in rat-liver mitochondria.

作者信息

Jamieson D, Chance B

出版信息

Biochem J. 1966 Jul;100(1):254-62. doi: 10.1042/bj1000254.

DOI:10.1042/bj1000254
PMID:5965254
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1265118/
Abstract
  1. The split-beam spectrophotometer was used to monitor changes in the steady state of cytochrome c and cytochromes a+a(3) during pressurization in pure oxygen. 2. High-pressure oxygen was found to cause oxidation of cytochrome c in rat-liver mitochondria, and of cytochromes a+a(3) at low pH. 3. No difference in these effects was found when various substrates were metabolized. 4. Lowering of pH markedly potentiated the high-pressure effect on the cytochromes. 5. Increased temperature and pressure hastened the reaction to high-pressure oxygen. 6. The oxidation of the cytochromes occurs on the substrate side of cytochrome c, probably at the dehydrogenase level, and the time-course of the reaction is compared with effects of oxygen toxicity in vivo.
摘要
  1. 采用分束分光光度计监测纯氧加压过程中细胞色素c和细胞色素a+a₃稳态的变化。2. 发现高压氧可导致大鼠肝脏线粒体中细胞色素c以及低pH值下细胞色素a+a₃的氧化。3. 当代谢各种底物时，未发现这些效应存在差异。4. pH值降低显著增强了高压对细胞色素的作用。5. 温度和压力升高加速了对高压氧的反应。6. 细胞色素的氧化发生在细胞色素c的底物侧，可能在脱氢酶水平，并且将反应的时间进程与体内氧毒性的效应进行了比较。