脂质过氧化抑制大鼠大脑皮质突触体ATP酶活性的可能机制。
Possible mechanism of inhibition by lipid peroxidation of ATPase activities of rat cerebral cortex synaptosomes.
作者信息
Sawas A H, Gilbert J C
出版信息
Arch Int Pharmacodyn Ther. 1984 May;269(1):4-11.
PMID:6087751
Abstract
The effects of metal ions and drugs which inhibit ATPase activities of synaptosomes have been determined on the lipid peroxidation activity of synaptosomes. Whilst some metal ions and drugs appear to influence the ATPase activities through a mechanism involving lipid peroxidation, others affect the enzymes directly or by other mechanisms.
摘要
已经测定了抑制突触体ATP酶活性的金属离子和药物对突触体脂质过氧化活性的影响。虽然一些金属离子和药物似乎通过涉及脂质过氧化的机制影响ATP酶活性，但其他一些则直接或通过其他机制影响这些酶。
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