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The action of palytoxin on erythrocytes and resealed ghosts. Formation of small, nonselective pores linked with Na+, K+-ATPase.

作者信息

Chhatwal G S, Hessler H J, Habermann E

出版信息

Naunyn Schmiedebergs Arch Pharmacol. 1983 Jul;323(3):261-8. doi: 10.1007/BF00497672.

DOI:10.1007/BF00497672
PMID:6137775
Abstract
摘要

相似文献

1
The action of palytoxin on erythrocytes and resealed ghosts. Formation of small, nonselective pores linked with Na+, K+-ATPase.岩沙海葵毒素对红细胞和重封血影的作用。与钠钾ATP酶相关的小的非选择性孔道的形成。
Naunyn Schmiedebergs Arch Pharmacol. 1983 Jul;323(3):261-8. doi: 10.1007/BF00497672.
2
Ouabain inhibits the increase due to palytoxin of cation permeability of erythrocytes.哇巴因抑制了由于岩沙海葵毒素导致的红细胞阳离子通透性增加。
Naunyn Schmiedebergs Arch Pharmacol. 1982 May;319(2):101-7. doi: 10.1007/BF00503920.
3
Palytoxin binds to and inhibits kidney and erythrocyte Na+, K+-ATPase.岩沙海葵毒素可结合并抑制肾脏和红细胞的钠钾ATP酶。
Naunyn Schmiedebergs Arch Pharmacol. 1984 Jan;325(1):85-7. doi: 10.1007/BF00507059.
4
Involvement of (Na+ + K+)-ATPase in binding and actions of palytoxin on human erythrocytes.(钠+钾)-ATP酶参与沙海葵毒素对人红细胞的结合及作用。
Biochim Biophys Acta. 1986 Sep 25;861(1):165-76. doi: 10.1016/0005-2736(86)90576-6.
5
Palytoxin promotes potassium outflow from erythrocytes, HeLa and bovine adrenomedullary cells through its interaction with Na+, K+ -ATPase.刺尾鱼毒素通过与钠钾ATP酶相互作用,促进钾离子从红细胞、人宫颈癌HeLa细胞和牛肾上腺髓质细胞中流出。
Toxicon. 1989;27(4):419-30. doi: 10.1016/0041-0101(89)90204-3.
6
Interaction of palytoxin with red cells: structure-function studies.岩沙海葵毒素与红细胞的相互作用:结构-功能研究
Toxicon. 1995 Jun;33(6):799-807. doi: 10.1016/0041-0101(95)00010-j.
7
Interaction of palytoxin and cardiac glycosides on erythrocyte membrane and (Na+ + K+) ATPase.岩沙海葵毒素与强心苷对红细胞膜及(钠+钾)ATP酶的相互作用。
Eur J Biochem. 1985 Oct 15;152(2):475-80. doi: 10.1111/j.1432-1033.1985.tb09221.x.
8
Sugar moiety of cardiac glycosides is essential for the inhibitory action on the palytoxin-induced K+ release from red blood cells.强心苷的糖部分对于抑制从红细胞中由岩沙海葵毒素诱导的钾离子释放的作用至关重要。
FEBS Lett. 1984 Jul 23;173(1):196-8. doi: 10.1016/0014-5793(84)81045-5.
9
Interactions of palytoxin with the Na,K-ATPase. Where are those sites?岩沙海葵毒素与钠钾ATP酶的相互作用。这些位点在哪里?
Ann N Y Acad Sci. 1997 Nov 3;834:424-5. doi: 10.1111/j.1749-6632.1997.tb52289.x.
10
Palytoxin acts through Na+,K+-ATPase.岩沙海葵毒素通过钠钾ATP酶发挥作用。
Toxicon. 1989;27(11):1171-87. doi: 10.1016/0041-0101(89)90026-3.

引用本文的文献

1
History and Toxinology of Palytoxins.Palytoxin 的历史与毒素学。
Toxins (Basel). 2024 Sep 26;16(10):417. doi: 10.3390/toxins16100417.
2
Palytoxin induces functional changes of anion transport in red blood cells: metabolic impact.海兔毒素诱导红细胞阴离子转运功能改变:代谢影响。
J Membr Biol. 2011 Jul;242(1):31-9. doi: 10.1007/s00232-011-9374-6. Epub 2011 Jul 6.
3
Large diameter of palytoxin-induced Na/K pump channels and modulation of palytoxin interaction by Na/K pump ligands.刺尾鱼毒素诱导的钠/钾泵通道的大直径以及钠/钾泵配体对刺尾鱼毒素相互作用的调节

本文引用的文献

1
The membrane locus of Ca-stimulated K transport in energy depleted human red blood cells.能量耗竭的人红细胞中钙刺激钾转运的膜位点。
J Membr Biol. 1971 Dec;6(4):315-28. doi: 10.1007/BF02116577.
2
The function of calcium in the potassium permeability of human erythrocytes.钙在人红细胞钾通透性中的作用。
Biochim Biophys Acta. 1958 Dec;30(3):653-4. doi: 10.1016/0006-3002(58)90124-0.
3
The interaction of potassium ions and ATP on the sodium pump of resealed red cell ghosts.钾离子与ATP对重新封闭的红细胞血影钠泵的相互作用。
J Gen Physiol. 2004 Apr;123(4):357-76. doi: 10.1085/jgp.200308964. Epub 2004 Mar 15.
4
Involvement of the Na,K-ATPase in the induction of ion channels by palytoxin.钠钾ATP酶参与岩沙海葵毒素诱导离子通道的过程。
Naunyn Schmiedebergs Arch Pharmacol. 1995 May;351(5):542-54. doi: 10.1007/BF00171047.
5
Palytoxin binds to and inhibits kidney and erythrocyte Na+, K+-ATPase.岩沙海葵毒素可结合并抑制肾脏和红细胞的钠钾ATP酶。
Naunyn Schmiedebergs Arch Pharmacol. 1984 Jan;325(1):85-7. doi: 10.1007/BF00507059.
6
Action and binding of palytoxin, as studied with brain membranes.用脑膜研究岩沙海葵毒素的作用与结合。
Naunyn Schmiedebergs Arch Pharmacol. 1983 Jul;323(3):269-75. doi: 10.1007/BF00497673.
7
Characterization of depolarization induced by palytoxin and grayanotoxin-I in isolated cardiac tissues from dogs and guinea pigs.海葵毒素和灰安妥毒素-I诱导的犬和豚鼠离体心脏组织去极化的特征
Naunyn Schmiedebergs Arch Pharmacol. 1985 Jul;330(1):67-73. doi: 10.1007/BF00586711.
8
Palytoxin induces a nonselective cation channel in single ventricular cells of rat.刺尾鱼毒素可在大鼠单个心室肌细胞中诱导形成非选择性阳离子通道。
Naunyn Schmiedebergs Arch Pharmacol. 1988 May;337(5):591-3. doi: 10.1007/BF00182738.
9
Single ionic channels induced by palytoxin in guinea-pig ventricular myocytes.海葵毒素在豚鼠心室肌细胞中诱导产生的单离子通道
Br J Pharmacol. 1988 Apr;93(4):811-6. doi: 10.1111/j.1476-5381.1988.tb11466.x.
10
Effects of palytoxin on guinea pig tracheal strips.岩沙海葵毒素对豚鼠气管条的作用。
Pharm Res. 1991 Jul;8(7):859-64. doi: 10.1023/a:1015895227196.
J Physiol. 1981;319:403-18. doi: 10.1113/jphysiol.1981.sp013917.
4
Preparation and properties of chromium(III) adenosine 5'-triphosphate, chromium(III) adenosine 5'-diphosphate, and related chromium(III) complexes.三价铬腺苷5'-三磷酸、三价铬腺苷5'-二磷酸及相关三价铬配合物的制备与性质
Biochemistry. 1980 Apr 1;19(7):1496-505. doi: 10.1021/bi00548a037.
5
Passive rubidium fluxes mediated by Na-K-ATPase reconstituted into phospholipid vesicles when ATP- and phosphate-free.当无ATP和磷酸盐时,由重构到磷脂囊泡中的钠钾ATP酶介导的被动铷通量。
J Physiol. 1982 Jul;328:295-316. doi: 10.1113/jphysiol.1982.sp014265.
6
Transport adenosine triphosphatases: properties and functions.转运ATP酶:特性与功能
Physiol Rev. 1981 Jan;61(1):1-76. doi: 10.1152/physrev.1981.61.1.1.
7
Inactivation of (Na+ + K+)-ATPase by chromium(III) complexes of nucleotide triphosphates.三磷酸核苷酸的铬(III)配合物对(钠+ +钾+)-ATP酶的失活作用。
Eur J Biochem. 1980 Aug;109(2):523-33. doi: 10.1111/j.1432-1033.1980.tb04824.x.
8
Inhibition by lead of human erythrocyte (Na+ + K+)-adenosine triphosphatase associated with binding of 210Pb to membrane fragments.
Biochim Biophys Acta. 1980 Jul 16;600(1):27-35. doi: 10.1016/0005-2736(80)90408-3.
9
Palytoxin both induces and inhibits the release of histamine from rat mast cells.岩沙海葵毒素既能诱导也能抑制大鼠肥大细胞释放组胺。
Int Arch Allergy Appl Immunol. 1982;68(2):97-100. doi: 10.1159/000233075.
10
Action and binding of palytoxin, as studied with brain membranes.用脑膜研究岩沙海葵毒素的作用与结合。
Naunyn Schmiedebergs Arch Pharmacol. 1983 Jul;323(3):269-75. doi: 10.1007/BF00497673.