Remesar X, Alemany M
Horm Metab Res. 1982 Apr;14(4):179-82. doi: 10.1055/s-2007-1018963.
Changes induced in liver and striated muscle glycogen and glycogen enzymes (glycogen synthetase, glycogen phosphorylase and alpha-amylase) by hypothyroidism and hyperthyroidism in rats have been determined. There were no changes in liver glycogen synthetase, phosphorylase and amylase activities in the hypothyroid group. Hyperthyroid rats showed lower liver glycogen synthetase, phosphorylase a and amylase activities. In muscle, hypothyroid rats had lower phosphorylase activity. In the hyperthyroid group glycogen synthetase was increased.--The results presented do not completely agree with the glycogen levels found in both tissues studied, and they are obviously more related to other factors such as glucose availability. It can be concluded that under the conditions studied, the glycogen enzyme levels could not alone explain the variations of glycogen levels.
已测定甲状腺功能减退和甲状腺功能亢进对大鼠肝脏和横纹肌糖原及糖原酶（糖原合成酶、糖原磷酸化酶和α-淀粉酶）的影响。甲状腺功能减退组大鼠肝脏糖原合成酶、磷酸化酶和淀粉酶活性无变化。甲状腺功能亢进的大鼠肝脏糖原合成酶、磷酸化酶a和淀粉酶活性较低。在肌肉中，甲状腺功能减退的大鼠磷酸化酶活性较低。甲状腺功能亢进组糖原合成酶增加。——所呈现的结果与在两个研究组织中发现的糖原水平并不完全一致，它们显然与其他因素如葡萄糖可用性更相关。可以得出结论，在所研究的条件下，糖原酶水平不能单独解释糖原水平的变化。
