Galbo H, Saugmann P, Richter E A
Acta Physiol Scand. 1979 Nov;107(3):269-72. doi: 10.1111/j.1748-1716.1979.tb06473.x.
Rats were either physically trained by a 12 wk swimming program or were freely eating or weight matched, sedentary controls. Trained rats had a higher relative liver weight and total hepatic glycogen synthetase (EC 2.4.1.11) activity and a lower phosphorylase (EC 2.4.1.1) activity than the other groups of rats. These changes may partly explain the demonstrated training-induced increase in glucose tolerance. None of the findings could be ascribed to differences in foold intake or body weight.
大鼠要么通过为期12周的游泳计划进行体能训练，要么作为自由进食或体重匹配的久坐对照组。与其他组大鼠相比，经过训练的大鼠相对肝脏重量更高，肝脏糖原合成酶（EC 2.4.1.11）总活性更高，磷酸化酶（EC 2.4.1.1）活性更低。这些变化可能部分解释了所证明的训练诱导的葡萄糖耐量增加。这些发现均不能归因于食物摄入量或体重的差异。
