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Role of substance P in the antidromic vasodilation, neurogenic plasma extravasation and disruption of the blood-aqueous barrier in the rabbit eye.

作者信息

Stjernschantz J, Sears M, Mishima H

出版信息

Naunyn Schmiedebergs Arch Pharmacol. 1982 Dec;321(4):329-35. doi: 10.1007/BF00498522.

DOI:10.1007/BF00498522
PMID:6188066
Abstract
摘要

相似文献

1
Role of substance P in the antidromic vasodilation, neurogenic plasma extravasation and disruption of the blood-aqueous barrier in the rabbit eye.P物质在兔眼逆行性血管舒张、神经源性血浆外渗及血-房水屏障破坏中的作用
Naunyn Schmiedebergs Arch Pharmacol. 1982 Dec;321(4):329-35. doi: 10.1007/BF00498522.
2
Substance P as neurogenic mediator of antidromic vasodilation and neurogenic plasma extravasation.P物质作为逆向性血管舒张和神经源性血浆外渗的神经源性介质。
Naunyn Schmiedebergs Arch Pharmacol. 1979 Dec;310(2):175-83. doi: 10.1007/BF00500282.
3
Effect of neurogenic irritation and calcitonin gene-related peptide (CGRP) on ocular blood flow in the rabbit.神经源性刺激和降钙素基因相关肽（CGRP）对兔眼血流的影响。
Curr Eye Res. 1988 Jul;7(7):695-703. doi: 10.3109/02713688809033199.
4
The effect of sodium iodate on the ciliary body/iris in rabbits.
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5
Effects of substance P on regional ocular blood flow, intraocular pressure and blood-aqueous barrier in rabbits.
Acta Ophthalmol (Copenh). 1989 Aug;67(4):378-82. doi: 10.1111/j.1755-3768.1989.tb01618.x.
6
Drug prevention of blood-aqueous barrier disruption.药物预防血-房水屏障破坏。
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7
Comparison of the effects of corticosteroids and indomethacin on the response of the blood-aqueous barrier to injury.
Curr Eye Res. 1987 Mar;6(3):419-25. doi: 10.3109/02713688709025197.
8
Studies on the trigeminal antidromic vasodilatation and plasma extravasation in the rat.大鼠三叉神经逆向性血管舒张和血浆外渗的研究。
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9
Indomethacin and the epinephrine-induced breakdown of the blood-ocular barrier in rabbits.
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10
Apraclonidine protection of the blood-aqueous barrier from traumatic break-down.阿可乐定对血-房水屏障免受外伤性破坏的保护作用。
J Ocul Pharmacol Ther. 1995 Spring;11(1):25-35. doi: 10.1089/jop.1995.11.25.

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Retinal laser burn-induced neuropathy leads to substance P-dependent loss of ocular immune privilege.视网膜激光灼伤诱导的神经病变导致 P 物质依赖性眼免疫特惠丧失。
J Immunol. 2012 Aug 1;189(3):1237-42. doi: 10.4049/jimmunol.1103264. Epub 2012 Jun 27.
2
Inhibitory effect of methysergide on calcitonin gene-related peptide-induced vasodilatation and ocular irritative changes in the rabbit.甲基麦角新碱对降钙素基因相关肽诱导的家兔血管舒张及眼部刺激性变化的抑制作用。
Br J Pharmacol. 1992 Jun;106(2):404-8. doi: 10.1111/j.1476-5381.1992.tb14347.x.

本文引用的文献

1
A HISTOCHEMICAL STUDY OF THE ADRENERGIC AND CHOLINERGIC INNERVATION OF THE ANTERIOR SEGMENT OF THE RABBIT EYE.兔眼前节肾上腺素能和胆碱能神经支配的组织化学研究
Invest Ophthalmol. 1964 Dec;3:592-600.
2
The distribution of mast cells in the normal eye. A method of study.正常眼内肥大细胞的分布。一种研究方法。
Exp Eye Res. 1963 Apr;2:134-40. doi: 10.1016/s0014-4835(63)80005-6.
3
Enzyme-linked immunosorbent assay of substance P: a study in the eye.P物质的酶联免疫吸附测定：眼部研究
J Neurochem. 1982 May;38(5):1323-8. doi: 10.1111/j.1471-4159.1982.tb07908.x.
4
A specific substance P antagonist blocks smooth muscle contractions induced by non-cholinergic, non-adrenergic nerve stimulation.一种特异性P物质拮抗剂可阻断由非胆碱能、非肾上腺素能神经刺激所引起的平滑肌收缩。
Nature. 1981 Dec 3;294(5840):467-9. doi: 10.1038/294467a0.
5
Time course of capsaicin-induced functional impairments in comparison with changes in neuronal substance P content.与神经肽P物质含量变化相比，辣椒素诱导的功能损伤的时间进程。
Naunyn Schmiedebergs Arch Pharmacol. 1981 Jun;316(3):240-3. doi: 10.1007/BF00505656.
6
Immunoreactivity for substance P in the Gasserian ganglion, ophthalmic nerve and anterior segment of the rabbit eye.兔眼三叉神经节、眼神经及眼前节中P物质的免疫反应性
Histochem J. 1981 May;13(3):435-43. doi: 10.1007/BF01005059.
7
Acetylcholinesterase hydrolyzes substance P.乙酰胆碱酯酶水解P物质。
Neuroscience. 1980;5(12):2065-72. doi: 10.1016/0306-4522(80)90124-4.
8
Intraocular effects of substance P in the rabbit.P物质对兔眼的眼内作用。
Invest Ophthalmol Vis Sci. 1981 Jan;20(1):53-60.
9
Substance P as a transmitter candidate.P物质作为一种潜在的神经递质。
Annu Rev Neurosci. 1980;3:227-68. doi: 10.1146/annurev.ne.03.030180.001303.
10
Substance P levels in normal and sensorily denervated rabbit eyes.正常及感觉神经去支配兔眼内的P物质水平。
Exp Eye Res. 1980 Mar;30(3):311-3. doi: 10.1016/0014-4835(80)90012-3.