Herold S, von Kummer R, Jaeger C
J Neurol. 1982;228(4):267-76. doi: 10.1007/BF00313417.
The follow-up of 89 patients with spontaneous intracerebral haematomas was studied by computed tomography (CT). The aetiology of the haemorrhage was found to be hypertension in 52 cases, vascular malformation in 14 cases, and two haematomas were due to anticoagulant therapy. The lesion visible on CT consisted of an area of increased density surrounded by a rim of low attenuation. Most cases showed a progressive decrease in size of the total lesion. Exceptions to this pattern were observed, which may lead to the misdiagnosis of a tumour. The central area of increased density decreased in size much more quickly than the total lesion. Signs of a mass effect were observed up to the ninth week in the larger lesions. The resorption of blood, measured by the decrease in diameter of the central high attenuation area, was not dependent on the site or original size of the haematoma, but was slower in hypertensive haemorrhage than in haemorrhage due to vascular malformation. Contrast enhancement was observed up to the ninth week and was ring-like in most cases. Our data confirm the well-known finding that the bleeding cannot be demonstrated by examination of the CSF if the blood has not reached the ventricular system or the subarachnoid space.
通过计算机断层扫描(CT)对89例自发性脑内血肿患者进行了随访研究。发现出血病因在52例中为高血压,14例为血管畸形,2例血肿是由于抗凝治疗所致。CT上可见的病变由密度增加区域被低衰减边缘围绕组成。大多数病例显示总病变大小逐渐减小。观察到有这种模式的例外情况,这可能导致误诊为肿瘤。密度增加的中心区域大小减小比总病变快得多。在较大病变中直至第九周都观察到占位效应的体征。通过中心高衰减区域直径减小来衡量的血液吸收,不取决于血肿的部位或原始大小,但高血压性出血比血管畸形所致出血吸收更慢。直至第九周都观察到对比增强,且大多数病例为环状。我们的数据证实了一个众所周知的发现,即如果血液未到达脑室系统或蛛网膜下腔,则通过脑脊液检查无法显示出血情况。