Baker R S, Buncic J R
Arch Neurol. 1984 Dec;41(12):1274-6. doi: 10.1001/archneur.1984.04050230060018.
A patient with a 16-month history of papilledema from pseudotumor cerebri had sudden loss of vision in one eye because of a central retinal artery occlusion (CRAO). The CRAO was transient and visual acuity improved from light perception to 6/7.5 OS in long-term follow-up. A visual field abnormality and color-vision deficit have persisted. Both eyes demonstrated peripapillary neovascular membranes. The eye with CRAO had extensive chorioretinal folds extending from the disc through the macula. Thorough investigations failed to demonstrate a coagulation abnormality, hyperviscosity, or source of emboli. Although the mechanism remains unclear, we postulate that CRAO may have been caused by a local pathologic condition in the optic nerve.
一名因假性脑瘤导致视乳头水肿16个月的患者，因视网膜中央动脉阻塞（CRAO）突然单眼失明。CRAO为一过性，长期随访中视力从光感改善至右眼6/7.5。视野异常和色觉缺陷持续存在。双眼均可见视乳头周围新生血管膜。发生CRAO的眼有广泛的脉络膜视网膜皱褶，从视盘延伸至黄斑。全面检查未发现凝血异常、高粘滞血症或栓子来源。尽管机制尚不清楚，但我们推测CRAO可能由视神经局部病理状况引起。
