Verma S R, Chand R, Tonk I P
Toxicol Lett. 1984 Jan;20(1):49-52. doi: 10.1016/0378-4274(84)90181-4.
Mercuric intoxication at sublethal levels (1/5, 1/10, 1/15, 1/20 or 1/25 fractions of 96 h LC50) produced alterations in the activity of serum glutamic oxalacetic transaminase (SGOT) and serum glutamic pyruvic transaminase (SGPT) of Notopterus notopterus. The difference between control and treated fish was found to be significant at P less than 0.05, P less than 0.01 and P less than 0.001 levels. Maximum (81.75%) significant (P less than 0.001) effect was observed in SGOT: maximum (69.23%) significant (P less than 0.001) effect was observed in SGPT in fish exposed for 60 days.
亚致死剂量（96小时半数致死浓度的1/5、1/10、1/15、1/20或1/25）的汞中毒导致长吻鱼血清谷氨酸草酰乙酸转氨酶（SGOT）和血清谷氨酸丙酮酸转氨酶（SGPT）的活性发生改变。在P小于0.05、P小于0.01和P小于0.001水平时，发现对照鱼和处理鱼之间的差异具有显著性。在暴露60天的鱼中，SGOT的最大显著效应（81.75%，P小于0.001）被观察到；SGPT的最大显著效应（69.23%，P小于0.001）被观察到。
