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Virus protein changes and RNA termini alterations evolving during persistent infection.

作者信息

Rowlands K, Grabau E, Spindler K, Jones C, Semler B, Holland J

出版信息

Cell. 1980 Apr;19(4):871-80. doi: 10.1016/0092-8674(80)90078-1.

DOI:10.1016/0092-8674(80)90078-1
PMID:6247073
Abstract
摘要

相似文献

1
Virus protein changes and RNA termini alterations evolving during persistent infection.在持续感染过程中发生的病毒蛋白变化和RNA末端改变。
Cell. 1980 Apr;19(4):871-80. doi: 10.1016/0092-8674(80)90078-1.
2
Gene expression of vesicular stomatitis virus genome RNA.水泡性口炎病毒基因组RNA的基因表达
Virology. 1992 Jun;188(2):417-28. doi: 10.1016/0042-6822(92)90495-b.
3
Vesicular stomatitis virus defective interfering particles can contain extensive genomic sequence rearrangements and base substitutions.水泡性口炎病毒缺陷干扰颗粒可包含广泛的基因组序列重排和碱基替换。
Cell. 1984 Apr;36(4):915-24. doi: 10.1016/0092-8674(84)90041-2.
4
The genetics of vesiculoviruses.水泡性病毒的遗传学
Arch Virol. 1982;72(1-2):1-34. doi: 10.1007/BF01314447.
5
Sequence-specific contacts between the RNA polymerase of vesicular stomatitis virus and the leader RNA gene.水疱性口炎病毒的RNA聚合酶与前导RNA基因之间的序列特异性接触。
Proc Natl Acad Sci U S A. 1981 Oct;78(10):6191-5. doi: 10.1073/pnas.78.10.6191.
6
Adding genes to the RNA genome of vesicular stomatitis virus: positional effects on stability of expression.向水疱性口炎病毒的RNA基因组中添加基因:对表达稳定性的位置效应。
J Virol. 2002 Aug;76(15):7642-50. doi: 10.1128/jvi.76.15.7642-7650.2002.
7
Sequence signal involved in the generation of an internally deleted defective interfering RNA from vesicular stomatitis virus.参与从水疱性口炎病毒产生内部缺失的缺陷干扰RNA的序列信号。
Nucleic Acids Res. 1982 Nov 11;10(21):6919-30. doi: 10.1093/nar/10.21.6919.
8
Cloning and expression of a viral phosphoprotein: structure suggests vesicular stomatitis virus NS may function by mimicking an RNA template.一种病毒磷蛋白的克隆与表达:结构表明水疱性口炎病毒NS可能通过模拟RNA模板发挥作用。
J Gen Virol. 1986 Aug;67 ( Pt 8):1571-9. doi: 10.1099/0022-1317-67-8-1571.
9
Preparation and analysis of the nucleocapsid proteins of vesicular stomatitis virus and sendai virus, and analysis of the sendai virus leader-NP gene region.水疱性口炎病毒和仙台病毒核衣壳蛋白的制备与分析,以及仙台病毒前导-NP基因区域的分析。
J Gen Virol. 1984 Apr;65 ( Pt 4):769-79. doi: 10.1099/0022-1317-65-4-769.
10
Frequent intragenic transcription termination within the N gene of vesicular stomatitis virus.水泡性口炎病毒N基因内频繁的基因内转录终止
Virology. 1982 Oct 30;122(2):239-50. doi: 10.1016/0042-6822(82)90224-0.

引用本文的文献

1
Defective interfering influenza virus confers only short-lived protection against influenza virus disease: evidence for a role for adaptive immunity in DI virus-mediated protection in vivo.缺陷干扰流感病毒只能提供短暂的流感病毒疾病保护:适应性免疫在体内 DI 病毒介导的保护中的作用证据。
Vaccine. 2011 Sep 2;29(38):6584-91. doi: 10.1016/j.vaccine.2011.06.114. Epub 2011 Jul 14.
2
Defective interfering particles of Sindbis virus do not interfere with the homologous virus obtained from persistently infected BHK cells but do interfere with Semliki Forest virus.辛德毕斯病毒的缺陷干扰颗粒不会干扰从持续感染的BHK细胞中获得的同源病毒,但会干扰Semliki森林病毒。
J Virol. 1981 Feb;37(2):840-4. doi: 10.1128/JVI.37.2.840-844.1981.
3
Evolution of virus and defective-interfering RNAs in BHK cells persistently infected with Sindbis virus.
辛德毕斯病毒持续感染的BHK细胞中病毒和缺陷干扰RNA的进化
J Virol. 1983 Dec;48(3):676-84. doi: 10.1128/JVI.48.3.676-684.1983.
4
Vesicular stomatitis virus mutants resistant to defective-interfering particles accumulate stable 5'-terminal and fewer 3'-terminal mutations in a stepwise manner.对缺陷干扰颗粒具有抗性的水疱性口炎病毒突变体以逐步方式积累稳定的5'-末端和较少的3'-末端突变。
J Virol. 1984 Mar;49(3):793-8. doi: 10.1128/JVI.49.3.793-798.1984.
5
Novel phenotype of RNA synthesis expressed by vesicular stomatitis virus isolated from persistent infection.从持续性感染中分离出的水疱性口炎病毒所表达的RNA合成新表型。
J Virol. 1982 Oct;44(1):167-74. doi: 10.1128/JVI.44.1.167-174.1982.
6
Enhanced mutability associated with a temperature-sensitive mutant of vesicular stomatitis virus.与水疱性口炎病毒温度敏感突变体相关的增强的变异性。
J Virol. 1981 Aug;39(2):377-89. doi: 10.1128/JVI.39.2.377-389.1981.
7
Viruses isolated from cells persistently infected with vesicular stomatitis virus show altered interactions with defective interfering particles.从持续感染水疱性口炎病毒的细胞中分离出的病毒,与缺陷干扰颗粒的相互作用发生了改变。
J Virol. 1980 Nov;36(2):627-31. doi: 10.1128/JVI.36.2.627-631.1980.
8
Persistent infection of some standard cell lines by lymphocytic choriomeningitis virus: transmission of infection by an intracellular agent.淋巴细胞性脉络丛脑膜炎病毒对某些标准细胞系的持续感染:一种细胞内因子介导的感染传播
J Virol. 1983 Oct;48(1):249-61. doi: 10.1128/JVI.48.1.249-261.1983.
9
The genetics of vesiculoviruses.水泡性病毒的遗传学
Arch Virol. 1982;72(1-2):1-34. doi: 10.1007/BF01314447.
10
Multiple viral mutations rather than host factors cause defective measles virus gene expression in a subacute sclerosing panencephalitis cell line.多种病毒突变而非宿主因素导致亚急性硬化性全脑炎细胞系中麻疹病毒基因表达缺陷。
J Virol. 1988 Apr;62(4):1388-97. doi: 10.1128/JVI.62.4.1388-1397.1988.