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Differential effects of retinoic acid and 7,8-benzoflavone on the induction of mouse skin tumors by the complete carcinogenesis process and by the initiation-promotion regimen.

作者信息

Verma A K, Conrad E A, Boutwell R K

出版信息

Cancer Res. 1982 Sep;42(9):3519-25.

PMID:6286109
Abstract
摘要

相似文献

1
Differential effects of retinoic acid and 7,8-benzoflavone on the induction of mouse skin tumors by the complete carcinogenesis process and by the initiation-promotion regimen.视黄酸和7,8-苯并黄酮对完整致癌过程及启动-促进方案诱导小鼠皮肤肿瘤的不同作用。
Cancer Res. 1982 Sep;42(9):3519-25.
2
The differential effects of retinoic acid and 7,8-benzoflavone on the induction of mouse skin tumors by the initiation-promotion protocol and by the complete carcinogenesis process.视黄酸和7,8-苯并黄酮通过启动-促进方案以及完整致癌过程对小鼠皮肤肿瘤诱导的差异效应。
Carcinog Compr Surv. 1982;7:35-9.
3
Inhibition of 7-bromomethylbenz[a]anthracene-promoted mouse skin tumor formation by retinoic acid and dexamethasone.维甲酸和地塞米松对7-溴甲基苯并[a]蒽促进的小鼠皮肤肿瘤形成的抑制作用。
Cancer Res. 1983 Jul;43(7):3045-9.
4
Vitamin A acid (retinoic acid), a potent inhibitor of 12-O-tetradecanoyl-phorbol-13-acetate-induced ornithine decarboxylase activity in mouse epidermis.维甲酸（视黄酸），一种对12 - O - 十四烷酰佛波醇 - 13 - 乙酸酯诱导的小鼠表皮鸟氨酸脱羧酶活性有强效抑制作用的物质。
Cancer Res. 1977 Jul;37(7 Pt 1):2196-201.
5
Is the initiation-promotion regimen in mouse skin relevant to complete carcinogenesis?小鼠皮肤中的启动-促进方案与完全致癌作用相关吗？
Carcinog Compr Surv. 1982;7:13-8.
6
Correlation of the inhibition by retinoids of tumor promoter-induced mouse epidermal ornithine decarboxylase activity and of skin tumor promotion.类视黄醇对肿瘤启动子诱导的小鼠表皮鸟氨酸脱羧酶活性的抑制作用与皮肤肿瘤促进作用的相关性。
Cancer Res. 1979 Feb;39(2 Pt 1):419-25.
7
[Effect of 7,8-benzoflavone on the incidence of skin tumors induced by polycyclic hydrocarbons].[7,8-苯并黄酮对多环烃诱导的皮肤肿瘤发生率的影响]
Biull Eksp Biol Med. 1979 May;87(5):454-5.
8
Inhibitory effect of 7,8-benzoflavone on DMBA- and BaP-induced bone marrow micronuclei in mouse.7,8-苯并黄酮对二甲基苯并蒽和苯并[a]芘诱导的小鼠骨髓微核的抑制作用。
Mutat Res. 1983 Aug;110(2):337-42. doi: 10.1016/0027-5107(83)90150-1.
9
Paradoxical effect of anthralin on 12-O-tetradecanoylphorbol-13-acetate-induced mouse epidermal ornithine decarboxylase activity, proliferation, and tumor promotion.蒽林对12-O-十四烷酰佛波醇-13-乙酸酯诱导的小鼠表皮鸟氨酸脱羧酶活性、增殖及肿瘤促进的矛盾效应。
Cancer Res. 1981 Jan;41(1):204-8.
10
Effects of 3-isobutyl-1-methylxanthine and cyclic nucleotides on the biochemical processes linked to skin tumor promotion by 12-O-tetradecanoylphorbol-13-acetate.3-异丁基-1-甲基黄嘌呤和环核苷酸对由12-O-十四烷酰佛波醇-13-乙酸酯介导的与皮肤肿瘤促进相关的生化过程的影响。
Cancer Res. 1981 Oct;41(10):3927-35.

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Profiling of the transcriptional response to all-trans retinoic acid in breast cancer cells reveals RARE-independent mechanisms of gene expression.全反式维甲酸诱导的乳腺癌细胞转录反应特征分析揭示了基因表达的 RARE 非依赖性机制。
Sci Rep. 2017 Nov 30;7(1):16684. doi: 10.1038/s41598-017-16687-6.
2
Association between cytoplasmic CRABP2, altered retinoic acid signaling, and poor prognosis in glioblastoma.细胞质CRABP2、视黄酸信号改变与胶质母细胞瘤预后不良之间的关联。
Glia. 2016 Jun;64(6):963-76. doi: 10.1002/glia.22976. Epub 2016 Feb 19.
3
CRABP-II- and FABP5-independent responsiveness of human glioblastoma cells to all-trans retinoic acid.
人胶质母细胞瘤细胞对全反式维甲酸的非CRABP-II和FABP5依赖性反应
Oncotarget. 2015 Mar 20;6(8):5889-902. doi: 10.18632/oncotarget.3334.
4
Curcumin restores sensitivity to retinoic acid in triple negative breast cancer cells.姜黄素可恢复三阴性乳腺癌细胞对维甲酸的敏感性。
BMC Cancer. 2014 Sep 27;14:724. doi: 10.1186/1471-2407-14-724.
5
Modulation of aryl hydrocarbon receptor (AHR)-dependent signaling by peroxisome proliferator-activated receptor β/δ (PPARβ/δ) in keratinocytes.过氧化物酶体增殖物激活受体β/δ（PPARβ/δ）对角质细胞中芳烃受体（AHR）依赖性信号的调节。
Carcinogenesis. 2014 Jul;35(7):1602-12. doi: 10.1093/carcin/bgu067. Epub 2014 Mar 17.
6
Cell survival signalling through PPARδ and arachidonic acid metabolites in neuroblastoma.过氧化物酶体增殖物激活受体 δ 和花生四烯酸代谢物在神经母细胞瘤中的细胞存活信号转导。
PLoS One. 2013 Jul 9;8(7):e68859. doi: 10.1371/journal.pone.0068859. Print 2013.
7
A mouse model for human anal cancer.人肛门癌的小鼠模型。
Cancer Prev Res (Phila). 2010 Dec;3(12):1534-41. doi: 10.1158/1940-6207.CAPR-10-0086. Epub 2010 Oct 6.
8
Overcoming retinoic acid-resistance of mammary carcinomas by diverting retinoic acid from PPARbeta/delta to RAR.通过将视黄酸从过氧化物酶体增殖物激活受体β/δ转移至维甲酸受体来克服乳腺癌的视黄酸抗性
Proc Natl Acad Sci U S A. 2008 May 27;105(21):7546-51. doi: 10.1073/pnas.0709981105. Epub 2008 May 21.
9
Is PPARbeta/delta a Retinoid Receptor?过氧化物酶体增殖物激活受体β/δ 是视黄酸受体吗？
PPAR Res. 2007;2007:73256. doi: 10.1155/2007/73256.
10
Opposing effects of retinoic acid on cell growth result from alternate activation of two different nuclear receptors.视黄酸对细胞生长的相反作用源于两种不同核受体的交替激活。
Cell. 2007 May 18;129(4):723-33. doi: 10.1016/j.cell.2007.02.050.