PLC/PRF/5人肝癌细胞的诱导性糖基化不足。简要报告。
Induced underglycosylation of PLC/PRF/5 human hepatoma cells. Brief report.
作者信息
Clementi M, Bagnarelli P, Pauri P, Brugia M
出版信息
Arch Virol. 1983;78(1-2):109-13. doi: 10.1007/BF01310864.
PMID:6316871
Abstract
The production of HBsAg and human plasma proteins by PLC/PRF/5 human hepatoma cell line was studied in the presence of glycosylation inhibitors. The data presented here suggest that HBsAg release in culture medium by these cells is not reduced by an inhibition of glycosylation and that Tunicamycin may play a role in a mobilization of the intracellular pool of antigen.
摘要
在存在糖基化抑制剂的情况下，对PLC/PRF/5人肝癌细胞系产生乙肝表面抗原（HBsAg）和人血浆蛋白的情况进行了研究。此处呈现的数据表明，这些细胞在培养基中释放HBsAg不会因糖基化受到抑制而减少，并且衣霉素可能在细胞内抗原池的动员中发挥作用。
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