Lucot J B
Brain Behav Evol. 1983;23(1-2):42-6. doi: 10.1159/000121487.
The evidence for possible roles of catecholamines in motion sickness is reviewed. l-Dopa is implicated in that it is the precursor to catecholaminergic neurotransmitters. Dopamine may have a role in stimulating motion sickness, though much basic information is lacking. Epinephrine may have a stimulatory role, since anatomical, pharmacological and physiological evidence is consistent with such a role. Norepinephrine probably inhibits motion sickness, as indicated by anatomical, physiological and pharmacological evidence. Information is missing that is critical to the understanding of the roles of each neurotransmitter in motion sickness.
本文综述了儿茶酚胺在晕动病中可能发挥作用的相关证据。左旋多巴与之相关，因为它是儿茶酚胺能神经递质的前体。多巴胺可能在引发晕动病中起作用，尽管目前仍缺乏许多基础信息。肾上腺素可能具有刺激作用，因为解剖学、药理学和生理学证据均支持这一作用。去甲肾上腺素可能抑制晕动病，解剖学、生理学和药理学证据均表明了这一点。对于理解每种神经递质在晕动病中的作用而言，关键信息仍有所缺失。
