Free-radical mechanisms in tissue injury.
作者信息
Slater T F
出版信息
Biochem J. 1984 Aug 15;222(1):1-15. doi: 10.1042/bj2220001.
Abstract
摘要
相似文献
1
Free-radical mechanisms in tissue injury.组织损伤中的自由基机制。
Biochem J. 1984 Aug 15;222(1):1-15. doi: 10.1042/bj2220001.
2
Free radicals and tissue injury: fact and fiction.自由基与组织损伤：事实与虚构
Br J Cancer Suppl. 1987 Jun;8:5-10.
3
Lipid peroxidation and cellular damage in toxic liver injury.中毒性肝损伤中的脂质过氧化与细胞损伤
Lab Invest. 1985 Dec;53(6):599-623.
4
Electron spin resonance study of homolytic cleavage of carbon tetrachloride in rat liver: pentadienyl free radicals.大鼠肝脏中四氯化碳均裂裂解的电子自旋共振研究：戊二烯基自由基
Pharmacol Res Commun. 1978 Jan;10(1):43-52. doi: 10.1016/s0031-6989(78)80061-7.
5
Pathological mechanisms in carbon tetrachloride hepatotoxicity.四氯化碳肝毒性的病理机制
J Free Radic Biol Med. 1985;1(1):27-38. doi: 10.1016/0748-5514(85)90026-1.
6
Carbon tetrachloride toxicity as a model for studying free-radical mediated liver injury.四氯化碳毒性作为研究自由基介导的肝损伤的模型。
Philos Trans R Soc Lond B Biol Sci. 1985 Dec 17;311(1152):633-45. doi: 10.1098/rstb.1985.0169.
7
[Oxygen radicals in biology].[生物学中的氧自由基]
Acta Med Port. 1984 Mar;5(3):103-7.
8
Free radicals and environmental toxins.自由基与环境毒素。
Ann Emerg Med. 1986 Sep;15(9):1075-83. doi: 10.1016/s0196-0644(86)80132-9.
9
The possible role of free radical reactions in carcinogenesis.自由基反应在致癌过程中的可能作用。
J Environ Pathol Toxicol. 1980 Mar;3(4 Spec No):273-303.
10
Hepatotoxicity of carbon tetrachloride in developing rats.四氯化碳对发育中大鼠的肝毒性。
Toxicol Appl Pharmacol. 1979 Sep 15;50(2):347-54. doi: 10.1016/0041-008x(79)90161-3.
引用本文的文献
1
Cell-Free DNA (cfDNA) Regulates Metabolic Remodeling in the ES-2 Ovarian Carcinoma Cell Line, Influencing Cell Proliferation, Quiescence, and Chemoresistance in a Cell-of-Origin-Specific Manner.游离DNA（cfDNA）调节ES-2卵巢癌细胞系中的代谢重塑，以细胞起源特异性方式影响细胞增殖、静止和化疗耐药性。
Metabolites. 2025 Apr 2;15(4):244. doi: 10.3390/metabo15040244.
2
Cell-Free DNA (cfDNA) Regulates Metabolic Remodeling, Sustaining Proliferation, Quiescence, and Migration in MDA-MB-231, a Triple-Negative Breast Carcinoma (TNBC) Cell Line.游离DNA（cfDNA）调节三阴性乳腺癌（TNBC）细胞系MDA-MB-231中的代谢重塑、维持增殖、静止和迁移。
Metabolites. 2025 Mar 27;15(4):227. doi: 10.3390/metabo15040227.
3
Antioxidants: a comprehensive review.抗氧化剂：全面综述。
Arch Toxicol. 2025 May;99(5):1893-1997. doi: 10.1007/s00204-025-03997-2. Epub 2025 Apr 15.
4
The effectiveness of antioxidant agents in delaying progression of diabetic nephropathy: A systematic review of randomized controlled trials.抗氧化剂在延缓糖尿病肾病进展中的有效性：随机对照试验的系统评价
Bioimpacts. 2024 Jun 19;15:30129. doi: 10.34172/bi.30129. eCollection 2025.
5
The J bs-5YP peptide can alleviate dementia in senile mice by restoring the transcription of Slc40a1 to secrete the excessive iron from brain.J bs-5YP肽可通过恢复Slc40a1的转录以分泌大脑中过量的铁来缓解老年小鼠的痴呆症状。
J Adv Res. 2025 Mar;69:51-59. doi: 10.1016/j.jare.2024.03.014. Epub 2024 Mar 23.
6
Integrative analysis identifies oxidative stress biomarkers in non-alcoholic fatty liver disease via machine learning and weighted gene co-expression network analysis.基于机器学习和加权基因共表达网络分析的整合分析确定非酒精性脂肪性肝病的氧化应激生物标志物。
Front Immunol. 2024 Feb 27;15:1335112. doi: 10.3389/fimmu.2024.1335112. eCollection 2024.
7
Coinfection with Enhances Disease Severity in Human African Trypanosomiasis.合并感染会加重人类非洲锥虫病的疾病严重程度。
J Trop Med. 2023 Nov 3;2023:1063169. doi: 10.1155/2023/1063169. eCollection 2023.
8
Ethyl Gallate Isolated from var. Branches Inhibits Melanogenesis and Promotes Autophagy in B16F10 Cells.从变种枝条中分离出的没食子酸乙酯抑制B16F10细胞的黑色素生成并促进自噬。
Antioxidants (Basel). 2023 Jan 25;12(2):269. doi: 10.3390/antiox12020269.
9
Chemotherapy-induced peripheral neuropathy in children and adolescent cancer patients.儿童和青少年癌症患者化疗引起的周围神经病变
Front Mol Biosci. 2022 Oct 14;9:1015746. doi: 10.3389/fmolb.2022.1015746. eCollection 2022.
10
The levels of oxidative stress in a combination of stress factors.在应激因素的组合中氧化应激的水平。
J Med Life. 2022 Aug;15(8):927-931. doi: 10.25122/jml-2021-0060.
本文引用的文献
1
Atmospheric carbon tetrachloride: another man-made pollutant.大气中的四氯化碳：另一种人为污染物。
Science. 1976 Jun 18;192(4245):1231-4. doi: 10.1126/science.192.4245.1231.
2
The effect of diet and 1,1,1-trichloro-2,2-bis-(p-chlorophenyl)ethane (DDT) on microsomal hydroxylating enzymes and on sensitivity of rats to carbon tetrachloride poisoning.饮食和 1,1,1-三氯-2,2-双（对氯苯基）乙烷（DDT）对微粒体羟化酶的影响以及对大鼠四氯化碳中毒敏感性的影响。
Biochem J. 1966 Aug;100(2):564-71. doi: 10.1042/bj1000564.
3
POSITIVE EVIDENCE OF ACCELERATION OF LIPOPEROXIDATION IN RAT LIVER BY CARBON TETRACHLORIDE: IN VITRO EXPERIMENTS.四氯化碳致大鼠肝脏脂质过氧化加速的阳性证据：体外实验
Life Sci (1962). 1965 Aug;4:1521-30. doi: 10.1016/0024-3205(65)90173-6.
4
METABOLISM OF CARBON TETRACHLORIDE AND CHLOROFORM BY THE RAT.大鼠对四氯化碳和氯仿的代谢
J Pharmacol Exp Ther. 1963 Aug;141:141-8.
5
6
NADPH-driven lipid peroxidation in rat liver nuclei and nuclear membranes.烟酰胺腺嘌呤二核苷酸磷酸（NADPH）驱动的大鼠肝细胞核及核膜脂质过氧化作用。
Arch Biochem Biophys. 1980 Mar;200(1):108-15. doi: 10.1016/0003-9861(80)90337-9.
7
Cytochrome b-245 of neutrophils is also present in human monocytes, macrophages and eosinophils.中性粒细胞的细胞色素b - 245也存在于人类单核细胞、巨噬细胞和嗜酸性粒细胞中。
Biochem J. 1981 Apr 15;196(1):363-7. doi: 10.1042/bj1960363.
8
Interactions of carbon tetrachloride and promethazine in the rat--II. Elimination of carbon tetrachloride and chloroform in expired air as indications of their metabolism in the intact animal.四氯化碳与异丙嗪在大鼠体内的相互作用——II. 呼出气体中四氯化碳和氯仿的消除作为其在完整动物体内代谢的指标
Biochem Pharmacol. 1981 Jun 15;30(12):1449-55. doi: 10.1016/0006-2952(81)90366-x.
9
Interactions of carbon tetrachloride and promethazine in the rat--I. Effects of promethazine on the concentrations of carbon tetrachloride in blood and liver, and on the production of chloroform.四氯化碳与异丙嗪在大鼠体内的相互作用——I. 异丙嗪对血液和肝脏中四氯化碳浓度以及氯仿生成的影响。
Biochem Pharmacol. 1981 Jun 15;30(12):1443-7. doi: 10.1016/0006-2952(81)90365-8.
10
Separation and characterization of the aldehydic products of lipid peroxidation stimulated by ADP-Fe2+ in rat liver microsomes.大鼠肝微粒体中由ADP - Fe2+刺激产生的脂质过氧化醛类产物的分离与鉴定
Biochem J. 1982 Oct 15;208(1):129-40. doi: 10.1042/bj2080129.
Zotero 插件