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核心技术专利：CN118964589B侵权必究

核心技术专利：CN118964589B侵权必究

Mills P R, Fell G S, Bessent R G, Nelson L M, Russell R I

Clin Sci (Lond). 1983 May;64(5):527-35. doi: 10.1042/cs0640527.

Zinc metabolism was studied in 11 patients with alcoholic cirrhosis and 13 healthy volunteers using the isotope tracer 65Zn. 2. Intestinal absorption, whole-body content and total daily elimination of zinc were all increased in alcoholic cirrhosis, but the biological half-life of zinc did not differ from controls. 3. Hepatic zinc concentration was reduced in alcoholic cirrhosis and correlated with changes in hepatic alcohol dehydrogenase activity. 4. Patients with alcoholic cirrhosis in this study were not demonstrated to be zinc-deficient.

使用同位素示踪剂65Zn对11例酒精性肝硬化患者和13名健康志愿者的锌代谢进行了研究。2. 酒精性肝硬化患者的肠道锌吸收、全身锌含量和每日锌总排泄量均增加，但锌的生物半衰期与对照组无差异。3. 酒精性肝硬化患者的肝脏锌浓度降低，且与肝脏乙醇脱氢酶活性的变化相关。4. 本研究中的酒精性肝硬化患者未表现出锌缺乏。

Mills P R, Fell G S, Bessent R G, Nelson L M, Russell R I

Clin Sci (Lond). 1983 May;64(5):527-35. doi: 10.1042/cs0640527.

Zinc metabolism was studied in 11 patients with alcoholic cirrhosis and 13 healthy volunteers using the isotope tracer 65Zn. 2. Intestinal absorption, whole-body content and total daily elimination of zinc were all increased in alcoholic cirrhosis, but the biological half-life of zinc did not differ from controls. 3. Hepatic zinc concentration was reduced in alcoholic cirrhosis and correlated with changes in hepatic alcohol dehydrogenase activity. 4. Patients with alcoholic cirrhosis in this study were not demonstrated to be zinc-deficient.

使用同位素示踪剂65Zn对11例酒精性肝硬化患者和13名健康志愿者的锌代谢进行了研究。2. 酒精性肝硬化患者的肠道锌吸收、全身锌含量和每日锌总排泄量均增加，但锌的生物半衰期与对照组无差异。3. 酒精性肝硬化患者的肝脏锌浓度降低，且与肝脏乙醇脱氢酶活性的变化相关。4. 本研究中的酒精性肝硬化患者未表现出锌缺乏。