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1
Resolution after radiotherapy of severe pulmonary damage due to paraquat poisoning.百草枯中毒所致严重肺损伤放疗后的转归
Br Med J (Clin Res Ed). 1984 Apr 28;288(6426):1259-60. doi: 10.1136/bmj.288.6426.1259.
2
Recovery after radiotherapy from severe interstitial pneumonia due to paraquat poisoning.百草枯中毒所致严重间质性肺炎放疗后的恢复情况。
Jpn J Med. 1987 Aug;26(3):385-7. doi: 10.2169/internalmedicine1962.26.385.
3
Pulmonary histological appearances in fatal paraquat poisoning.致命性百草枯中毒的肺部组织学表现。
Histopathology. 1978 Jan;2(1):53-66. doi: 10.1111/j.1365-2559.1978.tb01693.x.
4
Early radiotherapy in the treatment of paraquat poisoning.早期放射治疗在百草枯中毒治疗中的应用
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[Low-dose radiotherapy of pulmonary complications resulting from paraquat intoxication: report of a case].[低剂量放射治疗百草枯中毒所致肺部并发症:1例报告]
Taiwan Yi Xue Hui Za Zhi. 1987 Apr;86(4):452-5.
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Failure of radiotherapy to resolve fatal lung damage due to paraquat poisoning.放疗未能解决百草枯中毒所致的致命性肺损伤。
Chest. 1991 Oct;100(4):1164-5. doi: 10.1378/chest.100.4.1164.
7
Further clinical observations on the pulmonary effects of paraquat ingestion.关于百草枯摄入对肺部影响的进一步临床观察。
Thorax. 1979 Apr;34(2):161-5. doi: 10.1136/thx.34.2.161.
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[Effect of rhubarb as the main composition of sequential treatment in patients with acute paraquat poisoning: a prospective clinical research].[以大黄为主序贯治疗急性百草枯中毒患者的疗效:一项前瞻性临床研究]
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Ratio of angiopoietin-2 to angiopoietin-1 predicts mortality in acute lung injury induced by paraquat.血管生成素-2 与血管生成素-1 的比值预测百草枯诱导的急性肺损伤患者的死亡率。
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Paraquat intoxication in subjects who attempt suicide: why they chose paraquat.企图自杀者的百草枯中毒:他们为何选择百草枯。
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The area of ground glass opacities of the lungs as a predictive factor in acute paraquat intoxication.肺部磨玻璃影面积作为急性百草枯中毒的预测因素
J Korean Med Sci. 2009 Aug;24(4):636-40. doi: 10.3346/jkms.2009.24.4.636. Epub 2009 Jul 29.
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Comparison between kidney and hemoperfusion for paraquat elimination.肾脏与血液灌流清除百草枯的比较。
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Paraquat induced pulmonary fibrosis in three survivors.
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本文引用的文献

1
Recovery from severe paraquat poisoning following forced diuresis and immunosuppressive therapy.在进行强制利尿和免疫抑制治疗后从严重百草枯中毒中康复。
J Ir Med Assoc. 1971 Feb 4;64(405):69-71.
2
Experimental paraquat poisoning: a histological and electron-optical study of the changes in the lung.实验性百草枯中毒:肺脏变化的组织学和电子光学研究
J Pathol. 1971 Feb;103(2):123-9. doi: 10.1002/path.1711030207.
3
Pulmonary histological appearances in fatal paraquat poisoning.致命性百草枯中毒的肺部组织学表现。
Histopathology. 1978 Jan;2(1):53-66. doi: 10.1111/j.1365-2559.1978.tb01693.x.
4
Paraquat poisoning: significance of plasma-paraquat concentrations.百草枯中毒:血浆百草枯浓度的意义
Lancet. 1979 Aug 18;2(8138):330-2. doi: 10.1016/s0140-6736(79)90345-3.

百草枯中毒所致严重肺损伤放疗后的转归

Resolution after radiotherapy of severe pulmonary damage due to paraquat poisoning.

作者信息

Webb D B, Williams M V, Davies B H, James K W

出版信息

Br Med J (Clin Res Ed). 1984 Apr 28;288(6426):1259-60. doi: 10.1136/bmj.288.6426.1259.

DOI:10.1136/bmj.288.6426.1259
PMID:6424818
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1441111/
Abstract

A 29 year old man was admitted 36 hours after ingesting about 5 g paraquat. His arterial oxygen pressure fell progressively to 3.4 kPa (34 mm Hg), and pulmonary damage induced by paraquat was diagnosed. His condition did not improve after treatment with prednisolone and cyclophosphamide, but after irradiation both lungs cleared and arterial oxygen pressure started to improve. Irradiation of the lungs should be considered in patients who, after surviving the acute phase of poisoning with paraquat, show progressive deterioration of respiratory function.

摘要

一名29岁男子在摄入约5克百草枯36小时后入院。他的动脉血氧分压逐渐降至3.4 kPa(34 mmHg),诊断为百草枯所致的肺损伤。用泼尼松龙和环磷酰胺治疗后病情未改善,但双肺照射后病灶清除,动脉血氧分压开始改善。对于在百草枯中毒急性期存活后出现呼吸功能进行性恶化的患者,应考虑肺部照射。