缺氧引起的多巴胺代谢变化及促智药的作用

Biomed Biochim Acta. 1984;43(11):1301-4.

An 18 h lasting moderate hypoxia equivalent to 7000 m altitude rises DOPAC level in the caudate nucleus and the mesolimbic area of the rat to about 130%. This elevation is counteracted by a singular high dose of nootropics of the "energy mobilizer" type, but not by vasoactive ones.

持续18小时的相当于海拔7000米的中度缺氧会使大鼠尾状核和中脑边缘区域的3,4-二羟基苯乙酸（DOPAC）水平升高至约130%。这种升高可被一剂高剂量的“能量激活剂”类型的促智药抵消，但血管活性药物则无法抵消。