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Induction of experimental allergic encephalomyelitis in genetically resistant strains of mice.

作者信息

Lando Z, Teitelbaum D, Arnon R

出版信息

Nature. 1980 Oct 9;287(5782):551-2. doi: 10.1038/287551a0.

DOI:10.1038/287551a0
PMID:6448352
Abstract
摘要

相似文献

1
Induction of experimental allergic encephalomyelitis in genetically resistant strains of mice.在基因抗性小鼠品系中诱导实验性变应性脑脊髓炎
Nature. 1980 Oct 9;287(5782):551-2. doi: 10.1038/287551a0.
2
Effect of cyclophosphamide on suppressor cell activity in mice unresponsive to EAE.环磷酰胺对实验性自身免疫性脑脊髓炎无反应小鼠中抑制性细胞活性的影响。
J Immunol. 1979 Nov;123(5):2156-60.
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Glia cells as immunoregulatory elements. Up- and down-regulatory activities of astrocyte clones.
Ann N Y Acad Sci. 1988;540:463-5. doi: 10.1111/j.1749-6632.1988.tb27135.x.
4
Manipulation of autoimmune diseases with T-suppressor cells: lessons from experimental SLE and EAE.
Immunol Lett. 1993 May;36(2):109-16. doi: 10.1016/0165-2478(93)90041-y.
5
B lymphocytes treated in vitro with antigen coupled to cholera toxin B subunit induce antigen-specific Foxp3(+) regulatory T cells and protect against experimental autoimmune encephalomyelitis.体外经抗原耦联霍乱毒素 B 亚单位处理的 B 淋巴细胞诱导抗原特异性 Foxp3(+)调节性 T 细胞，并预防实验性自身免疫性脑脊髓炎。
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6
Transplantation of bone marrow transduced to express self-antigen establishes deletional tolerance and permanently remits autoimmune disease.转导以表达自身抗原的骨髓移植可建立缺失性耐受并永久性缓解自身免疫性疾病。
J Immunol. 2008 Dec 1;181(11):7571-80. doi: 10.4049/jimmunol.181.11.7571.
7
The thymus plays a role in oral tolerance in experimental autoimmune encephalomyelitis.胸腺在实验性自身免疫性脑脊髓炎的口服耐受中发挥作用。
J Immunol. 2006 Aug 1;177(3):1500-9. doi: 10.4049/jimmunol.177.3.1500.
8
Experimental autoimmune encephalomyelitis in mice: Lyt and Ia phenotypes of the effector and suppressor cells.小鼠实验性自身免疫性脑脊髓炎：效应细胞和抑制细胞的Lyt及Ia表型
Prog Clin Biol Res. 1984;146:277-84.
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Epidermal cells in activation of suppressor lymphocytes: further characterization.
J Immunol. 1987 Jun 15;138(12):4055-62.
10
Unresponsiveness to experimental allergic encephalomyelitis in mice: replacement of suppressor cells by a soluble factor.
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Life (Basel). 2022 Jun 27;12(7):962. doi: 10.3390/life12070962.
2
Resistance to oncolytic myxoma virus therapy in nf1(-/-)/trp53(-/-) syngeneic mouse glioma models is independent of anti-viral type-I interferon.NF1(-/-)/TRP53(-/-)同源性小鼠神经胶质瘤模型中对溶瘤性粘液瘤病毒治疗的耐药性与抗病毒 I 型干扰素无关。
PLoS One. 2013 Jun 6;8(6):e65801. doi: 10.1371/journal.pone.0065801. Print 2013.
3
G(i/o) protein-dependent and -independent actions of Pertussis Toxin (PTX).
百日咳毒素（PTX）的 G(i/o) 蛋白依赖性和非依赖性作用。
Toxins (Basel). 2011 Jul;3(7):884-99. doi: 10.3390/toxins3070884. Epub 2011 Jul 15.
4
Pertussis toxin up-regulates angiotensin type 1 receptors through Toll-like receptor 4-mediated Rac activation.百日咳毒素通过 Toll 样受体 4 介导的 Rac 激活上调血管紧张素 1 型受体。
J Biol Chem. 2010 May 14;285(20):15268-15277. doi: 10.1074/jbc.M109.076232. Epub 2010 Mar 15.
5
The postnatal maternal environment affects autoimmune disease susceptibility in A/J mice.产后母体环境影响 A/J 小鼠自身免疫性疾病的易感性。
Cell Immunol. 2010;260(2):119-27. doi: 10.1016/j.cellimm.2009.10.002.
6
Autoimmune thyroiditis: conversion of low-responder mice to high-responders by cyclophosphamide.自身免疫性甲状腺炎：环磷酰胺将低反应小鼠转化为高反应小鼠。
Clin Exp Immunol. 1982 Mar;47(3):683-8.
7
Promotion of spontaneous diabetes in non-obese diabetes-prone mice by cyclophosphamide.环磷酰胺促进非肥胖糖尿病易患小鼠自发性糖尿病的发生
Diabetologia. 1984 Dec;27(6):604-6. doi: 10.1007/BF00276978.
8
Resistance to the induction of EAE in AO rats: its prevention by the pre-treatment with cyclophosphamide or low dose of irradiation.AO大鼠对实验性自身免疫性脑脊髓炎诱导的抵抗：环磷酰胺预处理或低剂量照射对其的预防作用。
Clin Exp Immunol. 1982 Nov;50(2):311-7.
9
Semliki forest virus neurovirulence mutants have altered cytopathogenicity for central nervous system cells.
Infect Immun. 1982 Apr;36(1):333-41. doi: 10.1128/iai.36.1.333-341.1982.
10
Relapsing experimental allergic encephalomyelitis. An autoimmune model of multiple sclerosis.复发性实验性变应性脑脊髓炎。一种多发性硬化症的自身免疫模型。
Springer Semin Immunopathol. 1985;8(3):197-208. doi: 10.1007/BF00197296.