Mori E, Yamadori A, Kudo Y, Tabuchi M
Arch Neurol. 1984 Oct;41(10):1050-3. doi: 10.1001/archneur.1984.04050210048013.
We studied three cases that fit the clinical syndrome of ataxic hemiparesis. Computed tomography revealed small hemorrhages of the posterior limb of the internal capsule in all the cases, and somatosensory evoked potential studies predicted a disturbance of the lemniscal pathway from the thalamus to the parietal sensory cortex without demonstrable sensory loss. These results suggested that ataxia of the contralateral limbs following capsular lesion might result from a disruption of the thalamocortical sensory projection. The site of the lesion, the pathologic cause, and the underlying mechanism of ataxia are not necessarily homogeneous within the syndrome.
我们研究了三例符合共济失调性偏瘫临床综合征的病例。计算机断层扫描显示，所有病例的内囊后肢均有小出血灶，体感诱发电位研究预测从丘脑到顶叶感觉皮层的薄束通路存在障碍，但无明显感觉丧失。这些结果表明，囊病变后对侧肢体共济失调可能是由于丘脑皮质感觉投射中断所致。该综合征内病变部位、病理原因和共济失调的潜在机制不一定相同。
