Ortiz Camúñez M A, Martiń de Agar y Valverde M C, Lluch Fernández M D, Quesada Lucas M M, González Hachero J
An Esp Pediatr. 1984 Sep 15;21(3):191-7.
The role of sleep and sleep deprivation as inducer of paroxysmal abnormalities in EEG was studies in 104 infants and children under 14 years of age. These children suffered from various types of seizures and of normal standard EEG. These children who suffered from seizures of epileptic origin, after sleep deprivation had an EEG that showed a paroxysmal activity of 70.91% of the cases (p less than 0.001). The forced lack of sleep and the sleep that follows were found to be equally important as activators of paroxysmal abnormalities. The method of activation was found to be efficient in both partial an generalized seizures. The paroxysmal abnormalities were noted as being greater in phase II sleep. The EEG in sleep deprivation can show paroxysmal abnormalities, in children with epileptic seizures, that were hidden from standard EEG.
对104名14岁以下的婴幼儿及儿童进行了研究，以探讨睡眠和睡眠剥夺作为脑电图阵发性异常诱因的作用。这些儿童患有各种类型的癫痫发作且脑电图标准正常。这些患有癫痫源性发作的儿童，在睡眠剥夺后，脑电图显示70.91%的病例出现阵发性活动（p小于0.001）。发现强制睡眠剥夺及随后的睡眠作为阵发性异常的激活因素同样重要。这种激活方法在部分性发作和全身性发作中均有效。发现阵发性异常在睡眠二期更为明显。睡眠剥夺时的脑电图在患有癫痫发作的儿童中可显示出标准脑电图未显示的阵发性异常。
